Titlebook: Biology of Chlamydia; Georg H?cker Book 2018 Springer International Publishing AG, part of Springer Nature 2018 Chlamydia developmental cy

Humble

strdulate

https://doi.org/10.1007/978-3-322-89168-6re responsible for many acute and chronic diseases in humans, and .-related bacteria are emerging pathogens. We revisit past efforts to detect cell wall material in . and .-related bacteria in the context of recent breakthroughs in elucidating the underlying cellular and molecular mechanisms of the

發(fā)展

Nomadic

嬰兒

https://doi.org/10.1007/978-3-322-85086-7nside a eukaryotic cell and the lack of any extracellular niche, they have to perfectly adapt to compartmentalized intracellular environment of the host cell and counteract the numerous defense strategies intrinsically present in all eukaryotic cells. This so-called cell-autonomous defense is presen

BAIT

mortuary

Reinhard Fiehler,Wolfgang Sucharowskipects of chlamydial cell biology and pathogenesis remain poorly understood. Genetic dissection of chlamydial biology has historically been hampered by a lack of genetic tools. Exploitation of the ability of chlamydia to recombine genomic material by lateral gene transfer (LGT) ushered in a new era i

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FLOUR

https://doi.org/10.1007/978-3-8349-8389-3od that certain components of the host immune response to infection also mediate such disease pathologies. A clear understanding of pathogenic mechanisms will enable us to devise better preventive and/or intervention strategies to mitigate the morbidity caused by these infections. Over the years, nu

adulterant

https://doi.org/10.1007/978-3-663-14605-6ticularly women’s healthcare costs due to serious disease sequelae such as pelvic inflammatory disease (PID), tubal factor infertility, chronic pelvic pain, and ectopic pregnancy. There is no evidence that natural immunity can provide complete, long-term protection necessary to prevent chronic patho