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Titlebook: Biology of Brain Tumour; Proceedings of the S Michael D. Walker,David G. T. Thomas Conference proceedings 1986 Springer Science+Business Me

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發(fā)表于 2025-3-21 17:07:26 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Biology of Brain Tumour
期刊簡稱Proceedings of the S
影響因子2023Michael D. Walker,David G. T. Thomas
視頻videohttp://file.papertrans.cn/188/187684/187684.mp4
圖書封面Titlebook: Biology of Brain Tumour; Proceedings of the S Michael D. Walker,David G. T. Thomas Conference proceedings 1986 Springer Science+Business Me
影響因子This volume contains the proceedings of the Second International Symposium on Biology of Brain Tumour. The first Symposium was held in 1979 at Gardonne Riviera, Italy. This meeting was planned in order to coincide with the lOOth Anniversary of the first reported operation for glioma in London on November 25, 1884. Since the first meeting, the field of neuro-oncology has made remarkable progress in understanding both basic and clinical factors of significance to patients with brain tumor. While the earlier meeting dealt to a large extent with clinically oriented studies, this symposium was more heavily weighted toward the biology of brain tumour and improving our understanding at the physiologic, biochemical, pharmacologic, and cellular level. The meeting was divided according to scientific content into presentations and discussions as well as posters for more leisurely viewing, so as to allow the main themes of the meeting to sequentially develop. The first session dealt extensively with neuro-oncology at the molecular level and included considerable discus- sion of material related to the babic biochemical milieu in which tumors originate, proliferate, and eventually destroy the b
Pindex Conference proceedings 1986
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Two-dimensional gel electrophoretic protein patterns in high-grade human astrocytomasas and glioblastomas) obtained during surgery. Histological correlates of the sampled tissue were carefully established. There was a general consistency in the protein pattern from one sample to the next, despite variations in certain spot densities. When these fingerprints were compared to those of
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Dibutyryl adenosine 3′:5′-cyclic monophosphate (db-cAMP) induced growth inhibition and morphologic cthe human medulloblastoma cell line TE671 was responsive to this mechanism, cultured cells were treated with 0.01-1 mM concentrations of N.O.′-dibutyryl cyclic AMP (db-cAMP). Cell growth, morphologic changes, and expression of glial fibrillary acidic protein (GFAP) and neuron specific enolase (NSE)
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Neovascularisation of intracranial tumourslial-cell-stimulating angiogenesis factor (ESAF). The ex-tracts were separated into fractions of known molecular mass using gel-filtration chromatography. Neo-vascularisation resulting from angiogenic activity was assessed for each fraction using the chick chorioallan-toic (CAM) and chick vitelline
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