Titlebook: Biology of Aging; Alvaro Macieira-Coelho Book 2003 Springer-Verlag Berlin Heidelberg 2003 Alter.Cancer Research.Entwicklung.Geriatrics.Ger

Outwit

Marko Kr?tzig,Lutz Rauchhaupt,Darina Schulzeence is increasing at younger ages. More than half the cancers become clinically manifest during the second half of the human life span and their frequency increases with age, but their natural history starts way back at earlier ages. The data suggest that the late manifestation is the result of the

micronutrients

光亮

Neoplastic Growth Through the Human Life Span,ence is increasing at younger ages. More than half the cancers become clinically manifest during the second half of the human life span and their frequency increases with age, but their natural history starts way back at earlier ages. The data suggest that the late manifestation is the result of the

TOXIC

慢慢啃

Contort

Sedative

Aging of the Mammalian Organism,anism life span in a programmed and in a random fashion causing a modification of cell functioning and consequently, an evolution of cell interaction. Very little is known concerning the programmed evolution of the organism, the preordained changes through development, maturity, and involution of th

祖先

Neoplastic Growth Through the Human Life Span,ess with a linear increase in the incidence favored by aging of the organism. This is considered The Dogma and, with rare exceptions, another more realistic view of the problem is not accepted. The idea of a cancer-aging relationship is so ingrained that most authors describe only arguments favoring

Measured

The Centenarians,t 10 years from 112 to 122 years (Robine and Vaupel 2001). This is apparent when one examines the increase in the number of centenarians in indus trialized countries. Until about 1950, centenarians were quite rare (Table 9), since then their number has increased exponentially in low-mortality countr

粗野

9樓