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Titlebook: Biology and Management of Multiple Myeloma; James R. Berenson Book 2004 Springer Science+Business Media New York 2004 Staging.anemia.bone

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發(fā)表于 2025-3-21 19:41:45 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Biology and Management of Multiple Myeloma
影響因子2023James R. Berenson
視頻videohttp://file.papertrans.cn/188/187655/187655.mp4
發(fā)行地址Includes supplementary material:
學(xué)科分類Current Clinical Oncology
圖書封面Titlebook: Biology and Management of Multiple Myeloma;  James R. Berenson Book 2004 Springer Science+Business Media New York 2004 Staging.anemia.bone
影響因子Expert physicians and clinical researchers summarize and explain all the recent advances in the biology and treatment of bone marrow-based malignancy. On the biological side, the authors show the characteristics of the malignant cell and describe the significant roles played by oncogenic changes, chromosomal anomalies, Kaposi‘s sarcoma herpes virus, and cytokines. New epidemiological findings and prognostic factors are also analyzed. On the clinical side, the authors provide a comprehensive review of conventional treatment regimens, as well as a discussion of newer experimental approaches involving immunologic targeting, inhibitors of drug resistance, and antitumor agents.
Pindex Book 2004
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Dose-Intensive Therapy With Autologous Stem Cell Transplantation for Patients With Multiple Myelomah MM .. Barlogie and colleagues introduced the concept of high-dose chemotherapy followed by autologous stem cell transplantation in the 1980s . and rapidly several large trials were performed in patients with MM ..
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New Therapeutic Approaches to Myeloma,lan-prednisone (MP) combination therapy, a number of cytotoxic drug combinations have been investigated. A popular example is the M2 protocol, consisting of vincristine, carmustine, melphalan, cyclophosphamide, and prednisone (VBMCP) ..
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Sprechweisen in der (Konflikt)Moderationd that a less mature B lymphocyte or even a primordial stem cell might be clonal .. In this proposed model, these precursor cells could then sustain the plasma cell pool through differentiation in a manner akin to the process that occurs in chronic myelogenous leukemia. Recent advances in molecular
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Gesellschaftspolitik und Staatst?tigkeitdirect measures of tumor bulk and activity such as hemoglobin, calcium, serum and urine M-protein levels, number of bone lesions, and creatinine levels. Combining a series of inaccurate measures of tumor burden has proved to have limitations. In particular, quantification of bone lesions is unreliab
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