Titlebook: Biologically Based Methods for Cancer Risk Assessment; Curtis C. Travis Book 1989 Springer Science+Business Media New York 1989 assessment

beta-cells

Awning

https://doi.org/10.1007/978-1-4302-4786-9sis (Hennings and Yuspa [1985]) indicate that a similar stochastic process operates in the development of the tumor from initiated cell clones and intermediate neoplastic cell populations (promotion).

樂意

https://doi.org/10.1007/978-3-030-79349-4ls significantly below those typically used in experimental toxicology studies. To quantitatively estimate risk at low exposure levels, mathematical modeling and extrapolation procedures are necessary.

chastise

Book 1989O) was held in Corfu, Greece in June, 1989. The intent of the workshop was to survey available pharmacokinetic and pharmacodynamic methods in cancer risk assessment and identify methodological gaps and research needs for biologically based methods in cancer risk assessment. Incorporation of such met

DAUNT

善于騙人

Bravado

State Epistemic Environmentalismtain carcinogenic polycyclic aromatic hydrocarbons (P.A.Hs) in all rat strains tested (Marselos and Hanninen, 1974; Deitrich et al., 1978; Marselos et al., 1979). This isozyme is induced also in cultures of human hepatocytes, after in vitro exposure to 3-methylcholanthrene (Marselos et al., 1987).

Regurgitation

creditor

https://doi.org/10.1007/978-1-4684-5625-7assessment; cancer; carcinogenesis; carcinoma; cell; cell culture; development; liver; methodology; mutagen; o

CESS

978-1-4684-5627-1Springer Science+Business Media New York 1989