Titlebook: Biological, Diagnostic and Therapeutic Advances in Alzheimer‘s Disease; Non-Pharmacological Ghulam Md Ashraf,Athanasios Alexiou Book 2019

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,Genetics, Neuronal Pathways, and Electrophysiology of Alzheimer’s Disease,nsidered as the most common form of dementia, and it is likely to worsen over time; however, it is not a normal part of aging (Abraha et al. 2017). Dementia is associated with loss of memory and other cognitive capabilities of individuals. The individuals suffering from dementia have a comorbid heal

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,Neurobiological Mechanisms Involved in the Pathogenesis of Alzheimer’s Disease,accumulation of misprocessed and aggregated amyloid beta (Aβ)-plaques and appearance of intracellular neurofibrillary tangles containing hyperphosphorylated tau protein which ultimately leads to loss of synapses and cognitive decline. Aggregation of amyloid beta (Aβ)-plaques is the hallmark of AD. A

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,Immunotherapy in Alzheimer’s Disease, the imbalance between production and clearance of the toxic Aβ peptide (40–43 amino acid long) from the brain. Among all the therapeutic approaches, the most prominent one is immunotherapy. Both active and passive immunotherapies have been worked upon. The majority of active immunization-based meth

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Power and Powerlessness: What Is It?,function. Specifically, AD patients suffer from poor thinking skills and lack potential to perform simple activities. Dementia is multifactorial disorder and considered as the main cause of AD. Alzheimer’s disease is characterized by the presence of large quantities of two remarkable structures, i.e

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Henry J. Steadman,Eliot Hartstone is a complex and gradual process which involves degeneration of cells mainly due to cellular redox reactions that result in detrimental biochemical and genetic alterations. Studies have related the risk of age-related diseases with increased level of oxidants. Dietary polyphenols like flavonoids ar

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