Titlebook: Biological Response Modifiers in Human Oncology and Immunology; Thomas Klein,Steven Specter,Andor Szentivanyi Book 1983 The Editor(s) (if

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Corporeal

Michael K?lch,Paul L. Plener,J?rg M. Fegertmors, its utility has been limited due to toxicity to normal cells (2–3). Recent advances in experimental immunobiology, tumor immunology and cancer metastasis have increased our knowledge concerning the host’s immune response to tumors and the regulation of tumor growth (4–5). These advances have a

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Zwangsst?rungen im Kindes- und Jugendalter with primary and secondary immunodeficiency diseases (4). Thymus Factor X (TFX), an extract corresponding to thymosin fraction V prepared from calf thymus by Polfa Pharmaceuticals (Jelenia Góra, Poland) has been found to have similar immunomodulating properties (3). Thymic factors participate in th

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Zwangsst?rungen im Kindes- und Jugendalter the thymic hormones, thymosin (1), stimulates T cell development and corrects some immunodeficiency diseases resulting from lack of thymus functions. Considering the importance of T cells in immunoregulatory systems, thymosin is expected to be useful as a pharmaceutical agent for a variety of disea

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Tiefgreifende Entwicklungsst?rungeny of immunopathologic mechanisms. Autoantibodies and immune complexes may be present in the serum, and target organs may undergo vasculitic destruction or become infiltrated with lymphocytes and plasma cells. Recognition of histocompatibility antigens and Ia antigens on lymphocyte and macrophage mem

maintenance

L. Fr?lich,L. Hausner,F. Schneidermodulating host defense mechanisms in various ways. Table 1 summarizes the activities detected by in vivo assays. The in vivo activities include the modulation (mainly potentiation) of antibody- and cell-mediated immune responses and the stimulation of reticuloendothelial system. These activities po

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https://doi.org/10.1007/978-3-642-54571-9ignancy. Crude viable and/or killed mycobacterial agents are heterogenous preparations (2, 3), which have a significant incidence of morbidity. From an immunotherapeutic viewpoint, BCG has been shown (4, 5), in high doses, to cause splenic suppressor cells in mice, and has been implicated in causing