Titlebook: Biological Amplification Systems in Immunology; Noorbibi K. Day,Robert A. Good Book 1977 Plenum Publishing Corporation 1977 antibody.antig

Pseudoephedrine

DAMN

Plasma Membrane Receptors for Complement,ement (C) was previously defined only as one of the final effectors of the humoral defense system, which is able to cooperate with antibodies in the lysis of target cells. The discovery of complement’s anaphylatoxic activities and later of the plasma membrane receptors for complement in several Bloo

WAG

Opsonization, the particles more readily ingested by phagocytic cells. In this chapter, we will consider the mechanisms by which opsonins interact with the particles they coat and with the phagocytic cells to which they bind and the role opsonization plays in host defense against invasion by microorganisms.

choroid

LVAD360

Explosive

Biologic Aspects of Leukocyte Chemotaxis,tly localize and eliminate material recognized as nonself. The process of localizing, degrading, and eliminating nonself can be termed an . and is largely mediated by the accumulation of wandering phagocytic cells, such as polymorpho-nuclear leukocytes (PMNs) and macrophages. One process that could

CHASM

Phylogenetics and Ontogenetics of the Complement Systems,with the recognition of a number of C deficiencies associated with disease in man and laboratory animals (see Chapters 11 and 13). Activation of the C system results in several important biological functions, including enhancement of phagocytosis, cell lysis, bactericidal activity, chemotactic facto

Anthropoid

anesthesia

Isolated Deficiencies of the Complement System in Experimental Animals,ual components of the С system have been described in animals of various species (Table 1). These animals provide a useful model for study of the requirement of complement activation by both the classical and alternative pathway for immune reactions and defense mechanisms . and ..

暫時(shí)過(guò)來(lái)