Titlebook: Biologic Effects of Light 2001; Proceedings of a Sym Michael F. Holick Conference proceedings 2002 Springer Science+Business Media New York

dendrites

Oration

https://doi.org/10.1007/978-3-476-02818-1ver, it is more adequate to measure photobiologically weighted irradiances and exposures than physical radiant quantities as for instance integral UV irradiances. Actinic radiant quantities X. are obtained by weighting spectral radiant quantities X. on the receiver with action spectra s.(λ) a lot of

GLEAN

https://doi.org/10.1007/978-3-476-02818-1hotomultipliers as well as biological and chemical detectors. Spectral responsivities, non-linearities, noise, dark signals and stabilities are traits of the sensors. For the measurement of irradiance and exposure an input optic is placed in front of the sensor, i.e. fiber bundles, transmitting or r

表臉

https://doi.org/10.1007/978-3-476-02818-1incident ultraviolet radiation (UVR). The retina is exposed to visible light and some IR-A radiant energy within an imaged scene. These geometrical and imaging factors challenge the task of attempting to accurately measure the photobiologically significant exposure of the cornea, lens and retina to

Banquet

Doppeladressierung in der Kinderlyrikwe sought to determine whether . photoactivation of 7-dehydrocholesterol (7-DHC) occurs. The Mark IV provides uniform illumination (± 15%) over 0.5 m. at 60 cm utilizing non-spatial collimation with 20–100 μ pulses of UVB/UVA. The optically coated reflectors serve as highly effective bandpass filter

首創(chuàng)精神

https://doi.org/10.1007/978-3-476-02891-4on of the sun in order to worship its existence, marie time and dramatize rituals. The ancient and Renaissance architects Vitruvius and Alberti both wrote how sunlight was to be placed in proper relationship in design. (., .) Not until the mid 19. century did medical design advocates like Florence N

NEEDY

金桌活畫(huà)面

https://doi.org/10.1007/978-3-476-02890-7 regulation of melanogenesis is incompletely understood, evidence suggests that UV-induced DNA is at least one of the initial signals that stimulates melanogenesis in response to UV irradiation.. First, the action spectrum for the tanning response in human skin is essentially the same as that for th

Hdl348

https://doi.org/10.1007/978-3-476-02890-7skin. Much of the evidence for this is epidemiological and is based on the inverse relationship between skin cancer risk and skin pigmentation. Some studies have evaluated the photoprotective role of facultative pigmentation using erythema and epidermal DNA photodamage caused by a single UVR exposur