Titlebook: Biochemistry of Signal Transduction in Myocardium; Jos M. J. Lamers,Pieter D. Verdouw Book 1996 Kluwer Academic Publishers 1996 Angiotensi

Inveigle

書目名稱Biochemistry of Signal Transduction in Myocardium影響因子(影響力)




書目名稱Biochemistry of Signal Transduction in Myocardium影響因子(影響力)學(xué)科排名




書目名稱Biochemistry of Signal Transduction in Myocardium網(wǎng)絡(luò)公開度




書目名稱Biochemistry of Signal Transduction in Myocardium網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Biochemistry of Signal Transduction in Myocardium被引頻次




書目名稱Biochemistry of Signal Transduction in Myocardium被引頻次學(xué)科排名




書目名稱Biochemistry of Signal Transduction in Myocardium年度引用




書目名稱Biochemistry of Signal Transduction in Myocardium年度引用學(xué)科排名




書目名稱Biochemistry of Signal Transduction in Myocardium讀者反饋




書目名稱Biochemistry of Signal Transduction in Myocardium讀者反饋學(xué)科排名

嬰兒

Angiotensin II signalling pathways in cardiac fibroblasts: Conventional versus novel mechanisms in ml, and pathophysiological processes. Angiotensin II has been implicated in the developmental growth of the left ventricle in the neonate and remodeling of the heart following chronic hypertension and myocardial infarction. The inhibition of DNA synthesis and collagen deposition in myocardial interst

LEVER

Signalling by cGMP-dependent protein kinasescGMP are (i) phosphodiesterases, resulting in interference with the cAMP-signalling pathway, (ii) cGMP-gated cation channels, and (iii) cGMP-dependent protein kinases (cGKs). Only two mammalian isotypes of cGK have been described so far: type I cGK, consisting of an a and a β isoform, presumably spl

輕推

GEN

Regulation and functional significance of phospholipase D in myocardiumnative source of 1,2-diacylglycerol, which activates isoforms of protein kinase C. The members of the protein kinase C family respond differently to various combinations of Ca., phosphatidylserine, molecular species of 1,2-diacylglycerol and other membrane phospholipid metabolites including free fat

forbid

Mitogen-activated protein kinase (MAPK) in cardiac tissuestention being focussed on its possible role in the development of ventricular hypertrophy. It is becoming clear that MAPK is also active in other cells of cardiac origin such as cardiac fibroblasts and possible functions of this signalling pathway in the heart have yet to be explored. In this report

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Phosphotransfer reactions as a means of G protein activationymes or ion channels. Current models of G protein activation-deactivation indicate that the oligomeric GDP-bound form must undergo release of GDP, bind GTP and undergo subunit dissociation, in order to be in active form (GTP bound α subunits and free βγ dimers) and to regulate effectors. The effect

debunk

WAIL

fluoroscopy