Titlebook: Biochemistry of Inflammation; J. T. Whicher,S. W. Evans Book 1992 Springer Science+Business Media Dordrecht 1992 biochemistry.cytokine.inf

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書目名稱Biochemistry of Inflammation影響因子(影響力)




書目名稱Biochemistry of Inflammation影響因子(影響力)學(xué)科排名




書目名稱Biochemistry of Inflammation網(wǎng)絡(luò)公開度




書目名稱Biochemistry of Inflammation網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Biochemistry of Inflammation被引頻次




書目名稱Biochemistry of Inflammation被引頻次學(xué)科排名




書目名稱Biochemistry of Inflammation年度引用




書目名稱Biochemistry of Inflammation年度引用學(xué)科排名




書目名稱Biochemistry of Inflammation讀者反饋




書目名稱Biochemistry of Inflammation讀者反饋學(xué)科排名

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The kinin system: current concepts and perspectives,on of kinins is increased in the initial phase of the inflammatory process both in experimental animals. and in humans.. Despite their rapid inactivation by a variety of peptidases, a local generation of kinins may be maintained during the subacute and chronic phase of inflammation by the continuous

FILTH

Role of cellular proteinases and their protein inhibitors in inflammation, rubor, calor, and turgor are all dependent for their production on the action of proteolytic enzymes. There is no possibility of reviewing all of the proteinases involved in inflammation in a single chapter. There are literally scores of enzymes that would have to be considered. In order to reduce

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Eicosanoids (prostaglandins and leukotrienes),gues that aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) inhibit PG synthesis.. More recently, it has been suggested that other products of arachidonic acid metabolism, such as leukotrienes, are also mediators of allergic and inflammatory reactions. Inhibitors of leukotriene synthe

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chastise

Glucocorticoids and lipocortin,s this is predominantly hydrocortisone (cortisol), but other animals may release different but related steroids. For a long while it was believed that steroids were in some way involved in the mounting of the response to stress (such as an inflammatory response), but it has only recently become clea

mortuary

Platelet activating factor and cytokine feedback regulation of endothelial integrity; implications , while immune functions prevent invasion by foreign organisms. These two processes are closely linked as many of the same cells and molecules are operative in both. In a normal controlled response following injury, polymorphonuclear leukocytes (PMNL) leave the flowing blood and adhere to the endoth

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Tumour necrosis factor (TNF) and inflammation,, but nevertheless essential, aspect of these defences is the inflammatory response. In contrast to the immune responses involving antibodies and effector T lymphocytes, inflammation is relatively non-specific, but it does provide a quick and usually effective anti-invasive response. The inflammator

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