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Titlebook: Biochemistry of Copper; Maria C. Linder Book 1991 Springer Science+Business Media New York 1991 Absorption.biochemistry.chemistry.metaboli

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期刊全稱Biochemistry of Copper
影響因子2023Maria C. Linder
視頻videohttp://file.papertrans.cn/187/186703/186703.mp4
學(xué)科分類Biochemistry of the Elements
圖書封面Titlebook: Biochemistry of Copper;  Maria C. Linder Book 1991 Springer Science+Business Media New York 1991 Absorption.biochemistry.chemistry.metaboli
影響因子Copper has long been known as essential to living systems, in part through its fundamental role in electron transport and respiration. Over the years into the present, its involvement in an ever increasing number of processes in all kinds of organisms has become apparent, and new and exciting vistas of its roles in such areas as the central nervous system, and in humoral functions, are appearing on the horizon. Although the biochemistry of this element has not been studied nearly as much as that of many others, a for- midable amount of work has been carried out. It has thus been a challenge to produce a summary of what has been found that provides both breadth and depth. My goal has been to try to be as comprehensive as possible, within some limitations. I have tried to provide basic information and basic data that should continue to be useful for a long time. The goal has also been to interpret where we currently stand in our knowledge of the structure, function, regulation, and metabolism of Cu-dependent processes and sub- stances, especially proteins. Thus, I have tried to make this a source book for historic as well as current information on all aspects of copper bio- chemistry
Pindex Book 1991
The information of publication is updating

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Extracellular Copper Substituents and Mammalian Copper Transport, abundant ..-glycoprotein, long recognized as containing the largest proportion of copper in most animal and human plasma; albumin, containing (except in the dog, pig, and chicken) a single high-affinity Cu-binding site at the N-terminus; two larger, nonceruloplasmin, nonalbumin proteins, one called
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Copper and Disease,ve, and, if untreated, ultimately fatal disease of copper accumulation in the human body, particularly in the liver, brain, and kidney (see Table 9-1). Indeed, the demonstration of excess copper in the liver is a requisite for the diagnosis of Wilson’s disease. The disease is usually seen as hepatic
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