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Titlebook: Biochemical Pharmacology as an Approach to Gastrointestinal Disorders; Basic Science to Cli Timothy S. Gaginella,Gyula Mózsik,K. D. Rainsfo

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樓主: CLIP
21#
發(fā)表于 2025-3-25 03:47:15 | 只看該作者
22#
發(fā)表于 2025-3-25 11:27:57 | 只看該作者
23#
發(fā)表于 2025-3-25 14:09:17 | 只看該作者
Critical Evaluation of Acinar, Ductal, Vascular and Intestinal Intraluminal Factors Influencing Pancbe regarded as motives. Factors of defensive mechanism include prostaglandins, nitric oxide and unobstructed juice flow which promote the repair of injured membranes in acinar and vascular endothelial cells, respectively. Their whole sum may be called adaptive pancreatic cyto- and vasoprotection or pancreatic ‘self-defence mechanism’.
24#
發(fā)表于 2025-3-25 18:07:45 | 只看該作者
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發(fā)表于 2025-3-25 22:13:07 | 只看該作者
26#
發(fā)表于 2025-3-26 00:49:58 | 只看該作者
Leila Baradaran Jamili,Bahman Zarrinjooeest. As expected, both SP and VIP, microinjected into the nROb, evoked marked decreases in intragastric pressure before bicuculline. However, the gastric motor effects of SP, but not VIP, were abolished by bicuculline. Therefore we conclude that SP-evoked gastric relaxation in the nROb is mediated through a peripheral GABA-ergic pathway.
27#
發(fā)表于 2025-3-26 04:56:48 | 只看該作者
Mechanisms and Regulation of C1- Secretion in the Large Intestine: Studies with the Rat Distal ColonAMP-mediated secretion by intracellular cGMP, which affects Cl. secretion indirectly via a cGMP-sensitive phosphodiesterase, an enzyme which is responsible for cAMP degradation. These studies suggest a complex interaction of all three signalling pathways in the intracellular regulation of Cl. secretion.
28#
發(fā)表于 2025-3-26 08:32:29 | 只看該作者
29#
發(fā)表于 2025-3-26 13:53:54 | 只看該作者
30#
發(fā)表于 2025-3-26 17:53:24 | 只看該作者
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