Titlebook: Bile Acids in Gastroenterology; Basic and Clinical Susumu Tazuma,Hajime Takikawa Book 2017 Springer Japan KK 2017 Cholesterol.Colon Cancer.

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Bile Acids and Esophageal Cancerreactive oxygen species and activation of nuclear factor-κB and its downstream signaling pathways. Recent studies have revealed the characteristics of bile acid receptors and transporters in Barrett’s esophagus and esophageal adenocarcinoma.

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Bile Acids and NAFLD/NASHate bile acid biotransformation in the intestine, leading to alterations of lipid, glucose, and energy metabolism. This article reviews recent advances in the understanding of BAs signaling and its regulation of metabolic homeostasis in nonalcoholic fatty liver disease (NAFLD) and nonalcoholic steatohepatitis (NASH).

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Bile Acid and Colorectal Canceriferation, apoptosis, and oxidative DNA damage. On the other hand, certain bile acids, ursodeoxycholate, might protect against colorectal carcinogenesis. This review will examine the opposing effects on colorectal tumor promotion and tumor inhibition.

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Book 2017scussions on Bile Acid research and diseases. It begins with detailed discussions on the biological aspects of bile acid, with chapters examining bile acid from various perspectives, from its metabolism to its use in therapeutic agents. Subsequent chapters focus on diseases involving bile acid abnor

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Trennung von Isolaten aus Thomasroheisen, acids in humans. Neutral (classical) pathway is the major pathway of the bile acid biosynthesis in adult humans, which starts from 7ɑ-hydroxylation of cholesterol, catalyzed by cholesterol 7ɑ-hydroxylase (CYP7A1), the rate-limiting step of bile acid biosynthesis. In the liver, farnesoid X receptor