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Titlebook: Benign Hematologic Disorders in Children; A Clinical Guide Deepak M. Kamat,Melissa Frei-Jones Book 2021 Springer Nature Switzerland AG 2021

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樓主: 拿著錫
31#
發(fā)表于 2025-3-27 00:55:23 | 只看該作者
32#
發(fā)表于 2025-3-27 03:58:44 | 只看該作者
https://doi.org/10.1007/978-94-009-7439-5eficiency and pyruvate kinase (PK) deficiency. G6PD deficiency predominantly results in acute episodic hemolysis in response to exposure to oxidative stress. PK deficiency results in chronic non-spherocytic hemolytic anemia. Treatment of both disorders is supportive with a focus of avoiding offending agents in G6PD deficiency.
33#
發(fā)表于 2025-3-27 08:06:26 | 只看該作者
Vikram Kanagala M.D.,Daniel J. Stein M.D.iatric myeloproliferative disorder with distinct biological and clinical features when compared to adult essential thrombocytosis. In this chapter, we will provide an overview of primary and secondary causes of thrombocytosis.
34#
發(fā)表于 2025-3-27 12:54:09 | 只看該作者
35#
發(fā)表于 2025-3-27 14:50:20 | 只看該作者
36#
發(fā)表于 2025-3-27 20:15:01 | 只看該作者
37#
發(fā)表于 2025-3-28 01:39:01 | 只看該作者
Isabel Roitman,Anjali Mone,Arun Swaminathnticipatory guidance, prevention of bleeding complications, and screening for associated syndromic medical comorbidities. Treatment is generally supportive, and prognosis depends on the underlying cause.
38#
發(fā)表于 2025-3-28 05:16:26 | 只看該作者
https://doi.org/10.1007/978-3-319-75022-4Diagnosis lies in the careful assessment of the patient’s clinical features, and occasionally targeted genetic testing, as well as the timing and pattern of the neutropenia. Management varies from observation to use of colony-stimulating factors.
39#
發(fā)表于 2025-3-28 06:21:53 | 只看該作者
40#
發(fā)表于 2025-3-28 10:51:52 | 只看該作者
Thalassemiafelong chronic transfusions, leading to iron overload and risk for alloimmunization. New therapies are being investigated to decrease transfusion requirements; however, only hematopoietic stem cell transplant and recent gene therapy are able to completely eliminate the need for transfusions.
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