Titlebook: Beh?et Syndrome; Yusuf Yazici,Gulen Hatemi,Hasan Yazici Book 2020Latest edition Springer Nature Switzerland AG 2020 Beh?et’s disease.Beh?e

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縱火

https://doi.org/10.1007/978-3-030-38987-1munological characteristics of BS including the paradigm of anti-HLA autoimmunity. The α-tropomyosin model shares some clinical features of BS. This model has a potential to become a useful autoimmune model for BS. The only published trial to establish a transgenic model for BS did not show any sign

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Family Therapy: In and Out of SYNCut 10% of patients may become blind despite treatment. Similarly, prognosis is still grim for pulmonary artery aneurysms, which lead to death in about one fourth of the affected cases, and parenchymal neurological involvement, which results in death or severe disability in approximately half of the

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https://doi.org/10.1007/978-3-319-30954-5 BS and deciphering its yet unknown disease mechanisms. So instead of a universal, we should have subspecialty-based classification/diagnostic criteria sets. Similarly, we should more aim to understand how it differs from other inflammatory diseases of unknown etiology instead of going at lengths to

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Almar Otten,Arne Alphenaar,Han Witus stomatitis, genital ulcers, and various cutaneous lesions are its foremost manifestations. The presence of erythema nodosum-like lesions, pseudofolliculitis, papulopustular lesions, acneiform nodules, superficial thrombophlebitis, and cutaneous hyperreactivity (pathergy) are included in the crite

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