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Titlebook: Behavioral Neurobiology of Stress-related Disorders; Carmine M. Pariante,M. Danet Lapiz-Bluhm Book 2014 The Editor(s) (if applicable) and

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發(fā)表于 2025-3-21 16:10:27 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Behavioral Neurobiology of Stress-related Disorders
影響因子2023Carmine M. Pariante,M. Danet Lapiz-Bluhm
視頻videohttp://file.papertrans.cn/183/182864/182864.mp4
發(fā)行地址Includes clinical work on psychiatric disorders such as depression, anxiety and addiction.Final chapter with an outlook on resilience to stress.With a bench-to-bedside approach ?.Includes supplementar
學(xué)科分類Current Topics in Behavioral Neurosciences
圖書封面Titlebook: Behavioral Neurobiology of Stress-related Disorders;  Carmine M. Pariante,M. Danet Lapiz-Bluhm Book 2014 The Editor(s) (if applicable) and
影響因子Stress is such an over-used word that it is at time difficult to define its core features. When is an environment stressful? What does a stressful environment do to the brain and to the body? What are the biological mechanisms by which a stressor affects us? How does stress contributes to the onset and the progression of mental disorders? How do the effects of stress change over the life-time of an individual? These are just some of the overarching questions addressed by this book, thanks to the contribution of some of the world leading experts on the neurobiology of stress at the pre-clinical and clinical levels. Topics include current advances on the neurobiology of stress on various neurobiological systems such as immune, hypothalamic-pituitary-adrenal (HPA) axis, neurogenesis and neuroplasticity, neurotransmitter (glutamate, noradrenaline, dopamine, serotonin and endocannabinoid), neuropeptides, cognition and emotional processing as well as in utero and early postnatal effects. The clinical chapters deal with the relationship of stress and mental disorders such as depression, posttraumatic stress disorder (PTSD), anxiety disorders, schizophrenia, bipolar disorder, substance abu
Pindex Book 2014
The information of publication is updating

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發(fā)表于 2025-3-21 20:19:33 | 只看該作者
https://doi.org/10.1007/978-1-4842-6162-0the physical activity status of an organism may modulate stress-evoked neuronal-glial responses by changing the CX3CL1-CX3CR1 axis. Specifically, we propose that sedentary animals respond to an intense acute uncontrollable stressor with excessive serotonin (5-HT) and noradrenergic (NE) activity and/
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發(fā)表于 2025-3-22 05:35:45 | 只看該作者
Advanced Installation Techniqueson or methylation, which result in increased glucocorticoid transfer to the developing foetus, are linked to changes in offspring behaviour and in activity of the offspring HPA axis. Further understanding of the underlying pathways and identification of any gestation of vulnerability are needed to h
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發(fā)表于 2025-3-22 12:20:40 | 只看該作者
Pontus Cerin,Staffan LaestadiusTo date, fear conditioning and fear extinction animal models have provided support for the hypothesis that PTSD is a dysregulation of the processes related to fear regulation and, especially, fear extinction. More research is needed to further understand these processes as they relate not only to PT
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發(fā)表于 2025-3-22 14:15:00 | 只看該作者
Jaroslava Hyrslová,Miroslav Hájeksocial anxiety disorder have produced intriguing findings but not revealed a consistent pattern of endocrine disturbance, perhaps reflecting differences in methodology and the nature and size of the clinical samples. There is a persistent need for large, prospective studies using standardized method
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Tracy Dathe,Marc Helmold,Isabel Datheh as military personnel, relatively few randomized controlled trials have been conducted. Translational research, prospective longitudinal cohort studies, and clinical intervention trials are needed to better understand the behavioral neurobiology of stress and psychological resiliency.
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發(fā)表于 2025-3-23 01:31:08 | 只看該作者
Neuronal-Glial Mechanisms of Exercise-Evoked Stress Robustness,the physical activity status of an organism may modulate stress-evoked neuronal-glial responses by changing the CX3CL1-CX3CR1 axis. Specifically, we propose that sedentary animals respond to an intense acute uncontrollable stressor with excessive serotonin (5-HT) and noradrenergic (NE) activity and/
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