Titlebook: Behavioral Neurobiology of Depression and Its Treatment; Philip J. Cowen,Trevor Sharp,Jennifer Y. F. Lau Book 2013 The Editor(s) (if appli

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Genetic Mouse Models of Depression,amples of the use of complimentary lines, such as those that have genetic removal or overexpression, and also opposing behavioural changes seen following manipulation of closely related genes. Finally, factors such as the issue of background strain and influence of environmental factors are reflecte

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Inflammation and Depression,linical and animal studies, the role of stress, possible mechanisms and the involvement of genetic polymorphisms. Further understanding of pathways involved is still needed. This will be vital for the identification of new drug targets and preventative strategies.

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Emotional Processing and Antidepressant Action,lved in the later emergence of clinically relevant change. Such findings offer the possibility of greater integration between psychological and pharmacological conceptualisations of psychiatric illness and provide an experimental medicine model to generate and test specific predictions. Such a model

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Molecular and Cellular Mechanisms of Antidepressant Action,bic system are more easily remodelled by incoming emotionally relevant stimuli. This article attempts to bring together previous and current knowledge of antidepressant drug action on monoamine signalling at molecular and cellular levels, and introduces current thinking that these changes interact w

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https://doi.org/10.1007/978-3-8350-9085-9lt, several putative animal models of endophenotypes of depression or depression vulnerability have been proposed. Diathesis–stress models may aid in separating adaptive and maladaptive strategies in coping with stress, and understanding the relevant neurobiology. Studies comparing effects of stress