Titlebook: Behavioral Neurobiology of Bipolar Disorder and its Treatment; Husseini K. Manji,Carlos A. Zarate Jr. Book 2011 Springer-Verlag Berlin Hei

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書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment影響因子(影響力)




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment影響因子(影響力)學(xué)科排名




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment網(wǎng)絡(luò)公開度




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment被引頻次




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment被引頻次學(xué)科排名




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment年度引用




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書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment讀者反饋




書目名稱Behavioral Neurobiology of Bipolar Disorder and its Treatment讀者反饋學(xué)科排名

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https://doi.org/10.37307/b.978-3-503-19992-1 chromatin structure. In this chapter, we revisit the epidemiological, clinical, and molecular findings in BPD and reanalyze them from the perspective of inherited and acquired epigenetic misregulation. Epigenetic research has great potential to enhance our understanding of the molecular basis of BPD.

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https://doi.org/10.1007/978-3-322-93436-9on to many neurodegenerative disorders. It would be reasonable to assume that neurons regulating mood are progressively impaired during the course of BPD. Further studies are needed to clarify which neural systems are impaired by mitochondrial dysfunction in BPD.

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The Role of the Aminergic Systems in the Pathophysiology of Bipolar Disorder,the permissive hypothesis. Evidence from select studies of cerebrospinal fluid, postmortem subjects, neuroimaging, genetic factors, and pharmacological agents will be used to reconcile these hypotheses. Possible explanations for discrepancies in these hypotheses are given, and directions for future studies are suggested.

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Bipolar Disorder: A Neurobiological Synthesis,al impairment, psychosocial disability, and diminished well-being. Furthermore, the disorder is frequently unrecognized, misdiagnosed, and mismanaged, and much still remains unknown about its pathophysiology.

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