Titlebook: Behavioral Neurobiology of Alcohol Addiction; Wolfgang H. Sommer,Rainer Spanagel Book 2013 The Editor(s) (if applicable) and The Author(s)

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書(shū)目名稱Behavioral Neurobiology of Alcohol Addiction影響因子(影響力)




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書(shū)目名稱Behavioral Neurobiology of Alcohol Addiction被引頻次




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書(shū)目名稱Behavioral Neurobiology of Alcohol Addiction讀者反饋




書(shū)目名稱Behavioral Neurobiology of Alcohol Addiction讀者反饋學(xué)科排名

Myofibrils

Neurocircuitry Involved in the Development of Alcohol Addiction: The Dopamine System and its Accessand the possible involvement of acetaldehyde. Preclinical studies have provided the opportunity to dissect these interactions in some detail and although we do not fully comprehend the actions of ethanol there have been some great advances resulting in increased knowledge of the complexity of ethano

constellation

What is in that Drink: The Biological Actions of Ethanol, Acetaldehyde, and Salsolinol, interaction of the ethanol molecule with several neurochemical systems within the CNS. While there are scientific findings that offer support for both of these stances, the reinforcing properties of alcohol are most likely generated through a complex series of peripheral and central effects of both

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Modeling the Diagnostic Criteria for Alcohol Dependence with Genetic Animal Models,els in an attempt to determine whether they support a single-factor or multiple-factor genetic structure. We conclude that there is modest support in the animal literature that alcohol tolerance and withdrawal reflect distinct genetic risk factors, in agreement with our human data. We suggest areas

Slit-Lamp

Non-Human Primate Models of Alcohol-Related Phenotypes: The Influence of Genetic and Environmental nvironmental factors (i.e., stress, individual’s sex, or alcohol exposure) interact to influence alcohol consumption. Studies in primates may also reveal selective factors have driven maintenance or fixation of alleles that increase risk for alcohol use disorders in modern humans.

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Modeling Alcohol Self-Administration in the Human Laboratory,ere subjects press a button to request multiple sequential alcohol exposures intravenously instead of drinking. This method has been demonstrated to be safe and provides increased experimental control of BAC and keeps subjects blind concerning the amount already self-administered. Peak exposures in

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Animal Models of Excessive Alcohol Consumption in Rodents,rmittent access to alcohol; (d) scheduled-induced polydipsia; and (e) dependence and withdrawal experience. Each of the models possesses unique experimental features that engender excessive levels of alcohol consumption. Both advantages and disadvantages for each model are described along with discu

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Invertebrate Models of Alcoholism,odels should offer advantages over traditional model systems and should offer new insights into alcoholism-related behaviors. In this review we will summarize behavioral similarities and identified genes and mechanisms underlying ethanol-induced behaviors in invertebrates. This review mainly focuses