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Titlebook: Bacterial Infection: Close Encounters at the Host Pathogen Interface; Peter K. Vogt,Michael J. Mahan Book 1998 The Editor(s) (if applicabl

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發(fā)表于 2025-3-21 17:08:51 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱(chēng)Bacterial Infection: Close Encounters at the Host Pathogen Interface
影響因子2023Peter K. Vogt,Michael J. Mahan
視頻videohttp://file.papertrans.cn/181/180286/180286.mp4
學(xué)科分類(lèi)Current Topics in Microbiology and Immunology
圖書(shū)封面Titlebook: Bacterial Infection: Close Encounters at the Host Pathogen Interface; Peter K. Vogt,Michael J. Mahan Book 1998 The Editor(s) (if applicabl
影響因子When it comes to bacterial disease, we are living in a state of false security. Antibiotics have indeed brought unprecedented health benefits, protection from and cure of bacterial diseases during the past 50 years. But there are ominous signs that the fortress and the defenses built on antibiotics are crumbling. They are crum- bling because we wittingly or unwittingly created selective con- ditions for the emergence of superior pathogens that can no longer be controlled by antibiotics. There are numerous warnings. After a long period of eclipse tuberculosis has now emerged as a serious threat unchecked by antibiotic treatment. Recent years have seen reports of cholera epidemics, of anthrax infections, of serious problems with Salmonella and even with E. coli, just to name a few. Mankind is in a race with microbial invaders. The challenge is to anticipate and respond to developments that affect the precarious balance between man and microbe. This will re- quire new knowledge and it will take time for an effective appli- cation of that knowledge.
Pindex Book 1998
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New Insights into the Genetics and Regulation of Expression of , Enterotoxin,nary diseases (. et al. 1997). As their outstanding virulence attribute, these pathogenic Clostridia share the ability to elaborate extremely potent protein toxins (. et al. 1997). With the recent development of tools and techniques for analyzing clostridial genetics (. et al. 1997), it is now becom
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Mechanisms of Pathogenesis of Staphylococcal and Streptococcal Superantigens,cocci and streptococci, produce exoproteins which enhance microbial virulence. Among these proteins is the family of toxins known today as the superantigens (SAg). This family includes the pyrogenic toxin SAg (PTSAg), such as the staphylococcal enterotoxins (SE, serotypes A-E, G, H), group A strepto
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Intracellular Multiplication of ,: Human Pathogen or Accidental Tourist?,ses it has been possible to purify toxins as well as their receptors and targets. In some cases (e.g., diphtheria toxin, pertussis toxin), the basis of toxin activity is understood at the molecular level. For these organisms, it is possible to describe at least one important aspect of the pathogenes
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Identification of Virulence Determinants in Pathogenic Mycobacteria, the term “l(fā)epra” or “l(fā)epros” used in the Talmud and Old and New Testaments was different from the words used to describe leprosy in biblical times; “l(fā)epra” or “l(fā)epros” referred to specific scaling skin diseases such as psoriasis. However, the context in which the words “l(fā)epra” or “l(fā)epros” were used
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Jon Rahbek-Clemmensen,Line Jedig Nielsen the term “l(fā)epra” or “l(fā)epros” used in the Talmud and Old and New Testaments was different from the words used to describe leprosy in biblical times; “l(fā)epra” or “l(fā)epros” referred to specific scaling skin diseases such as psoriasis. However, the context in which the words “l(fā)epra” or “l(fā)epros” were used
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