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Titlebook: Atherosclerosis IV; Proceedings of the F G. Schettler,Y. Goto,G. Klose Conference proceedings 1977 Springer-Verlag Berlin Heidelberg 1977 A

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發(fā)表于 2025-3-21 18:15:17 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Atherosclerosis IV
期刊簡(jiǎn)稱Proceedings of the F
影響因子2023G. Schettler,Y. Goto,G. Klose
視頻videohttp://file.papertrans.cn/164/163949/163949.mp4
圖書封面Titlebook: Atherosclerosis IV; Proceedings of the F G. Schettler,Y. Goto,G. Klose Conference proceedings 1977 Springer-Verlag Berlin Heidelberg 1977 A
影響因子The presence of monotypism in thick atherosclerotic lesions of black females with G-6-PD mosaicism first reported by the Benditts (1973) has been confirmed in two other laboratories. However, we believe that it is premature to conclude that the finding of monotypism necessarily indicates monoclonal origin of athero- sclerotic lesions. We have suggested two alternative explanations for the obser- vation of monotypism which we believe must be shown to be invalid before accept- ing monoclonal origin as the only plausible way to account for the observed G-6-PD monotypism. One of these two alternatives relates to clonal heterogeneity of cell growth potential, i. e. , during the course of progressive growth of a le- sion, progeny of one cell may overgrow all others in a portion of the lesion. The other alternative is that one of the G-6-PD alleles may be linked to genes that afford a preferential survival characteristic in the abnormal environment present in atheroscerotic lesions. Thus, cells with one allele may be able to grow better than cells with the other allele, and this characteristic may be unrelated to "A-ness" or "B-ness". We have studied initiation of lesions in He diet-fed s
Pindex Conference proceedings 1977
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書目名稱Atherosclerosis IV影響因子(影響力)




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書目名稱Atherosclerosis IV網(wǎng)絡(luò)公開度學(xué)科排名




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Lipoproteins — Steady State Aspectsmunologically intact low-density lipoprotein (LP) is about the same as the concentration in the patients’ plasma, and in gelatinous thickenings, the intimai concentration of LP may be two or three times greater than the plasma concentration. In this talk, I will consider some of the factors that may
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The Kato decomposition property,those in the medium size arteries, such as the coronary and cerebral vessels; it is this ever-enlarging component of the lesions that is responsible for the encroachment upon the lumen and the ensuing consequences.
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Connective Tissue in Atherosclerosisthose in the medium size arteries, such as the coronary and cerebral vessels; it is this ever-enlarging component of the lesions that is responsible for the encroachment upon the lumen and the ensuing consequences.
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Arterial Endothelial Permeability to Macromoleculesristics,” and a possible regulatory control of medial metabolism. Each of these aspects deserves study and clarification. For example, lipoprotein lipase activity on or near the endothelial surface could have important implications not only in the conversion of VLDL to LDL (Fidge and Foxman, 1971),
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