Titlebook: Atherosclerosis; Cellular and Molecul Avrum I. Gotlieb,B. Lowell Langille,Sergey Fedorof Book 1991 Springer Science+Business Media New York

hypothyroidism

書目名稱Atherosclerosis影響因子(影響力)




書目名稱Atherosclerosis影響因子(影響力)學(xué)科排名




書目名稱Atherosclerosis網(wǎng)絡(luò)公開度




書目名稱Atherosclerosis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Atherosclerosis被引頻次




書目名稱Atherosclerosis被引頻次學(xué)科排名




書目名稱Atherosclerosis年度引用




書目名稱Atherosclerosis年度引用學(xué)科排名




書目名稱Atherosclerosis讀者反饋




書目名稱Atherosclerosis讀者反饋學(xué)科排名

tympanometry

魅力

Endothelial Cell — Extracellular Matrix Interactions: Modulation of Vascular Cell Phenotype by Matrito injury is less well-documented. Specifically, responses to injury by these vascular cell types appears to be modulated, in part, by the composition and organization of the surrounding matrix as well as by the various platelet factors and cytokines found at sites of injury. These observations sugg

eustachian-tube

Regulation of PDGF Expression in Vascular Cellsg proliferation of adjacent perivascular cells. If injury is sufficiently extensive, platelets can aggregate, degranulate and release PDGF. The growth factor deposited in these sites is chemotactic and mitogenic for vascular smooth muscle cells. The smooth muscle cells may also be activated to secre

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SEMI

poliosis

pathologists in the 1840‘s. Rudolf Virchow suggested that the movement of substances from the blood into the vessel wall was important for atherogenesis while Carl von Rokitansky felt that the deposition of substances on the lumenal surface of the artery resulted in the formation of atherosclerotic plaque. Si978-1-4613-6672-0978-1-4615-3754-0

記憶法

Schlüsseltexte aus der ersten Frauenbewegungg that heparin may act directly on SMC differentiation..Medial smooth muscle cells (SMC) proliferate and migrate towards the intima to form the bulk of the atheromatous plaque. Evaluation of cytoskeletal features of SMC during human early atheromatous plaques and rat experimental intimai thickenings

Synapse

DNR215