Titlebook: Asthma: Targeted Biological Therapies; Girolamo Pelaia,Alessandro Vatrella,Rosario Masell Book 2017 Springer International Publishing Swit

Anterior

Wilfried Pla?mann,Detlef Schulzr ability to induce type 2 innate lymphoid cells (ILC2s) to produce T2-type cytokines such as IL-5, IL-9 and IL-13 [2]. Therefore, IL-25, IL-33 and TSLP are suitable targets for antiasthma biological therapies [3].

誹謗

連鎖,連串

conflate

Airway Remodelling in Asthma,es which involve all layers of the bronchial wall, including large and small airways [1, 2]. Such structural changes, collectively referred as airway remodelling, likely originate from repetitive cycles of tissue injury, causing subsequent cellular responses leading to abnormal repair [3]. Although

eardrum

詞匯表

Lucubrate

Kinetic

,Anti-TNF-α Therapies, structural cells (Fig. 7.1) [1]. This pleiotropic cytokine is produced by several cell types including Th1 lymphocytes, macrophages and mast cells and induces the recruitment of neutrophils and eosinophils into the airways via up-regulation of epithelial and endothelial adhesion molecules [2, 3]. M

侵略

寵愛