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Titlebook: Eustachian Tube and Middle Ear Diseases; Peter A. Hall (Professor of Histopathology),David Book 1992 Springer-Verlag Tokyo 1992 communica

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發(fā)表于 2025-3-21 18:39:24 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Eustachian Tube and Middle Ear Diseases
影響因子2023Peter A. Hall (Professor of Histopathology),David
視頻videohttp://file.papertrans.cn/164/163375/163375.mp4
圖書封面Titlebook: Eustachian Tube and Middle Ear Diseases;  Peter A. Hall (Professor of Histopathology),David  Book 1992 Springer-Verlag Tokyo 1992 communica
影響因子For more than three decades the methods for assessing cell pro- liferation have been largely the preserve of experimental biologists, and in their hands such techniques have contributed greatly to our understanding of the dynamic organisation of normal and patho- logical tissues. In recent years, with the advent of novel method- ologies, there has been increased interest among both pathologists and clinicians, particularly oncologists and others interested in neoplasia, in assessing cell proliferation. This interest has been stimulated by the possibility that indices of cell proliferation may have direct clinical relevance, for example in being useful predictors of outcome in patients with certain forms of malignancy. In addi- tion, interest in assessing cell proliferation has been fuelled by the tremendous advances in our understanding of the mechanisms of cell proliferation and their deregulation in pathological processes. Consequently, the time is ripe for a monograph critically reviewing the available methods for assessing cell proliferation, their potential and their problems. We have been particularly concerned to present a balanced view of the advantages and disadvantages of
Pindex Book 1992
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Peter A. Hall (Professor of Histopathology),David
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Variation Within Ignitable Liquid Classes,rs primarily in G. (Smith 1982). Thus, though not invariant, the duration of S + G. + M generally changes far less than does the duration of G. with changes in proliferation rate. Furthermore, cells which cease proliferation, either reversibly (e.g. hepatocytes, lymphocytes) or permanently (e.g. neu
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Forensic Anthropology and Medicine these cells into highly ordered tissues with characteristic spatial and kinetic organisation (see for example Wright and Alison 1984). This is, in part, determined by the proliferative activity of progenitor cell populations within these tissues, but also by the number of growth-arrested cells, los
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Regulation of the Eukaryotic Cell Cycle,rs primarily in G. (Smith 1982). Thus, though not invariant, the duration of S + G. + M generally changes far less than does the duration of G. with changes in proliferation rate. Furthermore, cells which cease proliferation, either reversibly (e.g. hepatocytes, lymphocytes) or permanently (e.g. neu
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The Kinetic Organisation of Tissues, these cells into highly ordered tissues with characteristic spatial and kinetic organisation (see for example Wright and Alison 1984). This is, in part, determined by the proliferative activity of progenitor cell populations within these tissues, but also by the number of growth-arrested cells, los
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Measurement of Cell Proliferation Using Bromodeoxyuridine,to be true for chemotherapy when there are also intervals between treatments. In the case of radiotherapy, clinical trials are underway to assess strategies to overcome tumour cell proliferation during treatment by means of accelerated fractionation schedules when, in the extreme, the overall treatm
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Variation Within Ignitable Liquid Classes,ome condensation, nuclear envelope breakdown and spindle formation. With the advent of suitable radioactive tracers, it soon became clear that events relevant to cell division, notably DNA replication, actually began many hours before mitosis (Howard and Pelc 1953). This led to the subdivision of in
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