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Titlebook: Arterial Lesions and Arteriosclerosis; H. Jellinek (Professor of Pathology, Director) Book 1974 Akadémiai Kiadó, Budapest 1974 Lipoprotein

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樓主
發(fā)表于 2025-3-21 17:51:53 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Arterial Lesions and Arteriosclerosis
影響因子2023H. Jellinek (Professor of Pathology, Director)
視頻videohttp://file.papertrans.cn/162/161890/161890.mp4
圖書封面Titlebook: Arterial Lesions and Arteriosclerosis;  H. Jellinek (Professor of Pathology, Director) Book 1974 Akadémiai Kiadó, Budapest 1974 Lipoprotein
影響因子This monograph summarizes the results of a fifteen-year vascular pathology research project sponsored by the Hungarian Academy of Sciences. The workers who have participated in this project are indebted to the Academy for financing extensive investigations and visits to institutes abroad, the aims of which were to solve certain problems by international cooperation. The results presented by the collective effort of research teams, each studying here have been achieved a particular problem, but working together to the same end. Mutual discussion of findings and a regular exchange of information between the research groups have contributed to the progress of the investigations. Regular pUblication of details and mutual criticism ensured that the findings were uniformly interpreted. It is hoped that the fundamental aim of our long-term project, i. e. the selection of the right approach to the complex problem of vascular pathology, has been achieved. The new approach results from the recognition that since arteriosclerosis finally leads to various kinds of vascular injury, attention should be focussed on the events preceding the development of the lesions rather than on the lesions the
Pindex Book 1974
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沙發(fā)
發(fā)表于 2025-3-21 21:49:23 | 只看該作者
roach to the complex problem of vascular pathology, has been achieved. The new approach results from the recognition that since arteriosclerosis finally leads to various kinds of vascular injury, attention should be focussed on the events preceding the development of the lesions rather than on the lesions the978-1-4615-8713-2978-1-4615-8711-8
板凳
發(fā)表于 2025-3-22 02:13:06 | 只看該作者
地板
發(fā)表于 2025-3-22 06:18:41 | 只看該作者
Experimental Observations,scular layers as early as in 3–6 days and, in the meantime, the intensive intramural diffusion of plasma proteins (dyshoria) resulted in fibrinoid formation which first appeared confluent (Plate LXIII/47–50), then became circumferential (Plate LXIII/51). In the newly formed fibrinoid, single necroti
5#
發(fā)表于 2025-3-22 11:11:49 | 只看該作者
Conclusions from Available Experimental Data on the Role of Vascular Structure in the Morphology oflial fibrinoid. This change can be temporarily distinguished from the other form of fibrinoid change, which arises from necrotic smooth muscle cells of the media, hence the name media fibrinoid. Admixture of the two kinds of fibrinoid results in the characteristic lesion known generally as fibrinoid
6#
發(fā)表于 2025-3-22 14:17:22 | 只看該作者
https://doi.org/10.1007/978-3-663-07167-9om different kinds of injury are usually so similar that the tissue changes in themselves are as a rule not conclusive as to the nature of the causative factor. The phenomena of vascular injury are essentially uniform, but they may be modified by the duration of the damage, the structure of the vess
7#
發(fā)表于 2025-3-22 17:18:45 | 只看該作者
Alexander Nikuradse,Karl Kuglerscular layers as early as in 3–6 days and, in the meantime, the intensive intramural diffusion of plasma proteins (dyshoria) resulted in fibrinoid formation which first appeared confluent (Plate LXIII/47–50), then became circumferential (Plate LXIII/51). In the newly formed fibrinoid, single necroti
8#
發(fā)表于 2025-3-22 23:27:30 | 只看該作者
9#
發(fā)表于 2025-3-23 04:55:52 | 只看該作者
,Herstellung des Frischm?rtels,the findings were uniformly interpreted. It is hoped that the fundamental aim of our long-term project, i.e. the selection of the right approach to the complex problem of vascular pathology, has been achieved.
10#
發(fā)表于 2025-3-23 08:01:47 | 只看該作者
,Herstellung des Frischm?rtels,egments served as control. In addition to the usual staining techniques, some formalin-fixed specimens were stained with Carere-Coombs’ Siena-orange technique, to demonstrate smooth muscle cells which stain specifically yellow owing to the reaction of their potassium contents.
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