Titlebook: Arrhythmogenic RV Cardiomyopathy/Dysplasia; Recent Advances Frank I. Markus,Andrea Nava,Gaetano Thiene Book 20071st edition Springer-Verlag

使迷惑

Genotype-Phenotype Correlations,asing number of subjects, providing better knowledge of the diagnostic criteria, natural history, and ethiopathogenesis of the disease. Three different groups of genes have been found to be linked to ARVC/D: the ryanodine receptor-2 gene (RyR2), the gene encoding for the growth factor TGFbeta 3 and

修正案

Autopsy and Endomyocardial Biopsy Findings,racterized by progressive fibrofatty replacement of right ventricular myocardium, initially with typical regional and later global right and some left ventricular involvement, with relative sparing of the septum [.]. The residual myocytes interspersed among adipocytes and fibrous tissue provides the

壓迫

Coterminous

Dysplasia

鄙視讀作

Possible Causative or Contributing Role of Viruses, of the right ventricle and clinically by life-threatening ventricular arrhythmias in apparently healthy people [.]. The disease results in a high risk of sudden death, particularly in young people [.] and athletes [.]. Over the past several years, the genetic basis of this disease has been unravele

CERE

保全

多骨

瑣碎

Electrocardiographic Manifestations,cardiomyopathy/dysplasia (ARVC/D) [.–.]. Typically, ARVC/D is considered in a young or middle-aged individual with a history of ventricular arrhythmias who does not have evidence of ischemic heart disease. The suspicion of ARVC/D is increased if the standard 12-lead ECG shows features suggestive of