Titlebook: Arachidonic Acid Metabolism and Tumor Promotion; Susan M. Fischer,Thomas J. Slaga Book 1985 Martinus Nijhoff Publishing, Boston 1985 Nucle

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書目名稱Arachidonic Acid Metabolism and Tumor Promotion影響因子(影響力)




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書目名稱Arachidonic Acid Metabolism and Tumor Promotion被引頻次學(xué)科排名




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語(yǔ)言學(xué)

Arachidonic Acid Metabolism in the Skin,ught about by rapid biosynthesis in response to a variety of chemical, physical and hormonal factors. This synthesis or arachldonlc acid cascade is complex and includes several enzymes that are affected by endogenous and/or exogenous regulatory factors. Generally, molecular oxygen is inserted into a

劇本

Arachidonic Acid Metabolism and Tumor Promotion, modulatory role in the development of neoplasias. Mouse skin has proved to be one of the best model systems for studying the multistage nature of carcinogenesis (1–3). Skin tumors in mice can be readily induced by the sequential application of a subthreshold dose of a carcinogen (initiation stage)

GLUT

Prostaglandins, Epidermal Hyperplasia and Skin Tumor Promotion,nated arachidonic acid derivatives such as hydroxy-, hydroperoxy fatty acids and leukotrienes, prostaglandins have been reported as being produced in many tissues (for review see 1,2). Generally, production in a given tissue is initiated by an exogenous or endogenous stimulus and the prostaglandins

畢業(yè)典禮

FEAS

Tumor Promoting Phorbol Esters May Affect Cell Membrane Signal Transmission and Arachidonate Metabod influence growth and differentiation of cells in culture (5,6,7,8). These studies have taken a major step forward with the identification and characterization of a receptor protein to which phorbol esters bind. These findings, especially aspects relevant to arachidonate metabolism, are reviewed be

CREST

Active Oxygen and Promotion,ms of active oxygen are superoxide (O.) and its conjugate acid the hydroperoxyradical (HO.), singlet oxygen (O.), the hydroxyl radical (.OH) and hydrogenperoxide (H.O.). On the basis of epidemiological considerations, Totter (1) has proposed that oxygen-induced cellular damage rather than exposure t

旁觀者

Possible Involvement of Arachidonate Products in Tumor Promoter Inhibition of Cell-Cell Communicatih the environment via different kinds of feedback or communicating mechanisms (1,2). Unless there is a functional hierarchical organization of the molecular, biochemical, cellular, physiological and psychological (in the case of human beings), this state of health cannot be maintained. Proper geneti

明智的人

Proteases and Cyclic Nucleotides,function of general protein degradation, they also produce limited proteolysis which converts zymogens to active enzymes by the hydrolysis of a single peptide bond or of several bonds by sequential action. Much work over the past 10–15 years has been directed at the determination of a specific role

ONYM