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Titlebook: Approaches to Chronic Kidney Disease; A Guide for Primary Jerry McCauley,Seyed Mehrdad Hamrahian,Omar H. Maa Book 2022 Springer Nature Swi

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發(fā)表于 2025-3-28 14:42:25 | 只看該作者
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發(fā)表于 2025-3-28 19:49:37 | 只看該作者
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發(fā)表于 2025-3-28 22:59:19 | 只看該作者
Progression of CKD and Uremic Symptoms,mptomatic. Chronic interstitial inflammation, fibrosis, and tubular atrophy are the main pathological findings. Inflammatory markers play a major role in the pathogenesis of CKD along with chronic renin-angiotensin-aldosterone system (RAAS) activation.
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發(fā)表于 2025-3-29 04:47:46 | 只看該作者
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發(fā)表于 2025-3-29 08:15:33 | 只看該作者
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發(fā)表于 2025-3-29 12:25:39 | 只看該作者
Book 2022ditions and most common co-morbidities such as diabetes and hypertension. The chapters, that follow focus on the CKD associated complications and the CKD within special populations such as the elderly and minorities as well as dietary restrictions and drug dosing. The book concludes with discussion
47#
發(fā)表于 2025-3-29 17:42:40 | 只看該作者
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發(fā)表于 2025-3-29 20:32:09 | 只看該作者
Slowing Chronic Kidney Disease Progression,ression (e.g., estimated GFR <30?mL/min/1.73 m. or rapid decline in estimated GFR, albuminuria ≥300 mg per 24?hours) and be promptly referred to a nephrologist. Management of CKD mainly includes cardiovascular risk reduction (e.g., blood pressure control and statin), reduction of albuminuria (e.g.,
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發(fā)表于 2025-3-30 02:06:36 | 只看該作者
Hepatorenal Syndrome,tually renal function impairment. The diagnosis of HRS is that of exclusion, relying on serum creatinine levels and responses to aggressive volume expansion. Sometimes, it is difficult to differentiate HRS from acute tubular necrosis (ATN). Once the diagnosis is clear, diuretics should be discontinu
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發(fā)表于 2025-3-30 05:50:42 | 只看該作者
Complications of Chronic Kidney Disease: Electrolyte and Acid-Base Disorders,um bicarbonate until the estimated glomerular filtration rate (eGFR) falls to a value of less than 25?mL per minute per 1.73 m. (CRIC). This is not an absolute as it can be seen at all levels of CKD. Both a high anion gap metabolic acidosis and a non-gap acidosis can be found in patients suffering f
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