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Titlebook: Apoptosis; Enrico Mihich,Robert T. Schimke Book 1994 Springer Science+Business Media New York 1994 Antigen.DNA.apoptosis.cancer.cell.cell

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51#
發(fā)表于 2025-3-30 09:05:45 | 只看該作者
Regulation of Apoptosis by the Transforming Gene Products of Adenovirus,nslation, and DNA replication machinery to reproduce viral proteins and DNA. In rodent cells which are semipermissive for adenovirus infection, cell growth is deregulated but virus replication is ineffective. As the viral infection does not progress to completion, the deregulation of cell growth con
52#
發(fā)表于 2025-3-30 15:07:51 | 只看該作者
,: Oncogene and Tumour Suppressor Gene,s underscores a major problem facing multicellular organisms: namely, how to allow the rapid proliferation of component cells and at the same time never allow those component cells to engage in competition with each other. In principle, any cell that acquires a growth advantage through mutation shou
53#
發(fā)表于 2025-3-30 17:46:08 | 只看該作者
Regulation of Apoptosis in Interleukin-3-Dependent Myeloid Progenitor Cells,in hemopoietins.. To examine signalling pathways which may influence apoptosis in hematopoietic progenitors we have utilized the Interleukin-3 (IL-3) dependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for de
54#
發(fā)表于 2025-3-31 00:43:04 | 只看該作者
55#
發(fā)表于 2025-3-31 01:05:35 | 只看該作者
Molecular Controls of Cell Cycle Progression Following DNA Damage: Roles of P53 and Ataxia-Telangiere, an understanding of the molecular events involved in the cellular responses to such exposures should provide insights into mechanisms of human carcinogenesis. Much effort over the past 25 years has been focused on how altered nucleotide bases are removed and how the linear integrity of the DNA s
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