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Titlebook: Apoptosis; Y. Kuchino,W. E. G. Müller Book 1996 Springer-Verlag Berlin Heidelberg 1996 HIV.Krebs.Thymus.Viruses.Zelltod.apoptosis.cell.cel

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發(fā)表于 2025-3-21 17:42:48 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Apoptosis
影響因子2023Y. Kuchino,W. E. G. Müller
視頻videohttp://file.papertrans.cn/159/158993/158993.mp4
學科分類Progress in Molecular and Subcellular Biology
圖書封面Titlebook: Apoptosis;  Y. Kuchino,W. E. G. Müller Book 1996 Springer-Verlag Berlin Heidelberg 1996 HIV.Krebs.Thymus.Viruses.Zelltod.apoptosis.cell.cel
影響因子Apoptosis plays a central role in the regulation of cell proliferation. Disruption of this control mechanism may cause serious human diseases such as encephalomyelitis and cancer. Thus, understanding of the molecular mechanisms of apoptotic cell death should lead to fundamental advances in the therapy of these diseases..
Pindex Book 1996
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書目名稱Apoptosis影響因子(影響力)




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,: Antidote for Cell Death,ong oncogenes in its ability to enhance cell survival by interfering with apoptotic cell death. This finding provided the important notion to the cancer research field that inhibition of cell death might be a critical step in tumorigenesis. This idea was supported by the demonstration that several c
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Myc-Mediated Apoptosis,n testis, respectively. Our recent findings demonstrated that s-Myc expression suppresses the growth activity and tumorigenicity of glioma cells, indicating that s-Myc acts as a negative regulator in tumor growth. In addition, we found that s-Myc overexpression can effectively induce apoptotic cell
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Clusterin: A Role in Cell Survival in the Face of Apoptosis?,and proposed functions include the transport of lipoproteins, the inhibition of complementmediated cell lysis and the modulation of cell-cell interactions. On the basis of its elevated expression in apoptotic tissues, it was originally proposed that the protein might be casually involved in apoptosi
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Progress in Molecular and Subcellular Biologyhttp://image.papertrans.cn/a/image/158993.jpg
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