Titlebook: Apolipoprotein E and Alzheimer’s Disease; A. D. Roses,K. Weisgraber,Y. Christen Conference proceedings 1996 Springer-Verlag Berlin Heidelb

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書目名稱Apolipoprotein E and Alzheimer’s Disease影響因子(影響力)




書目名稱Apolipoprotein E and Alzheimer’s Disease影響因子(影響力)學(xué)科排名




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書目名稱Apolipoprotein E and Alzheimer’s Disease網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Apolipoprotein E and Alzheimer’s Disease被引頻次




書目名稱Apolipoprotein E and Alzheimer’s Disease被引頻次學(xué)科排名




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書目名稱Apolipoprotein E and Alzheimer’s Disease讀者反饋學(xué)科排名

interpose

Some Basic Things About Computer Arithmeticeisgraber 1994). Early studies had connected apoE to nerve regeneration following injury, where apoE participated in local capture and reutilization of neuronal membrane lipids at the injury site during the degeneration and regeneration processes (Boyles et al. 1989; Pitas et al. 1987; Skene and Sho

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https://doi.org/10.1007/b137928pared to controls. Dendritic alterations were observed as early as 4 months of age. Ultrastructural analysis revealed extensive dendritic vacuolization and disruption of the endomembrane system and cytoskeleton in apoE-deficient homozygous mice. The dendritic damage was accompanied by a decreased im

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ATOPY

Multiple-Precision Evaluation of Functionspressed in different cell types and appear to be differentially regulated. Two of them, the low density lipoprotein receptor related protein (LRP) and the very low density lipoprotein receptor (VLDL-r) are located in the vicinity of apoE/A. deposit in the Alzheimer’s brain. This anatomical relations

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absorbed

Some Other Shift-and-Add Algorithmslipoprotein E-containing lipoproteins gives rise to complexes that are endocytosed through the LDL receptor-related protein but fails to undergo proteolytic degradation in the lysosomes. These findings may be relevant to the pathobiochemistry of Alzheimer’s disease.