Titlebook: Aplastic Anemia; Pathophysiology and Hermann Heimpel,Edward C. Gordon-Smith,Bernhard Ku Book 1979 Springer-Verlag Berlin Heidelberg 1979 A

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https://doi.org/10.1007/978-3-662-11652-4Some, but not all patients with idiopathic acquired aplastic anemia get long lasting remissions and are potentielly cured after treatment with antilymphocyte globulin (ALG) [10, 11]. This suggests that the diagnosis covers disorders of various origin. Since the pathogenesis is unknown, prediction of response to ALG is impossible.

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Epidemiology and Aetiology of Aplastic AnemiaEpidemiological findings are of great importance as they may help us to understand aetiology better. My considerations of the epidemiology of aplastic anemia will be divided into two parts, the first being an analysis of the differences in prevalence of aplastic anemia between East and West, the second a short summary of a Swedish study.

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Aplastic Anemia — A Stem Cell Disorder?The pathogenesis and often the etiology of aplastic anemia is still obscure [1, 4, 10]. However, it is generally recognised that there is a basic defect in the pluripotent stem cell compartment which results in a diminished feeding of later, more differentiated compartments, eventually leading to a failure of the production of mature cells.

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Estimation of the Pluripotential Stem Cells in Bone Marrow After HydroxyureaThe method available at present for the measurement of the pluripotential stem cells (CFU.) is based on the formation of the macroscopically visible colonies on the spleen surface of the lethally irradiated mice recipients of the bone marrow cell suspension [9]. This method demonstrates a fraction of the pluripotential stem cells injected [7].

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Independent Requirements for Early and Late Stages of Erythropoiesisth in culture even in the presence of albumin, transferrin and lipids. On the basis of these observations, it was suggested that the action of erythropoietin might be confined to later stages of erythroid maturation subsequent to BFU-E [3, 6, 7].