Titlebook: Anxiety and Anxiolytic Drugs; Florian Holsboer,Andreas Str?hle Book 2005 Springer-Verlag Berlin Heidelberg 2005 Anxiety.Anxiolytic Drugs.N

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Mutagenesis and Knockout Models: NK1 and Substance P,substance P (SP) and its cognate receptor neurokinin 1 (NK1R) seem to play a particularly important role in higher brain functions. They are expressed at high levels in the limbic system, which is the neural basis of emotional responses. Three different lines of evidence from physiological studies s

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Genetic Epidemiology of Anxiety Disorders,ty disorder, and obsessive—compulsive disorder. Controlled family studies reveal that all of these anxiety subtypes are familial, and twin studies suggest that the familial aggregation is attributable in part to genetic factors. Panic disorder and, its spectrum have the strongest magnitude of famili

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Interactions Between Corticotropin-Releasing Hormone and Serotonin: Implications for the Aetiology panic disorder, has been increasing steadily during the past several years. Although the picture is far from complete yet—partly due to the large number of serotonin (5-HT) receptors and the often-disparate effects of receptor agonists and antagonists in animal models of anxiety—SSRIs and the 5-HT.

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Neuropeptides in Anxiety Modulation,e as endogenous modulators of complex behaviours, including anxiety-related behaviour and psychopathology, particularly due to their high number and diversity, the dynamics of release patterns in distinct brain areas and the multiple and variable modes of interneuronal communication they are involve