Titlebook: Antiarrhythmic Drugs; Mechanisms of Antiar Günter Breithardt,Martin Borggrefe,Gerhard Hindric Book 1995 Springer-Verlag Berlin Heidelberg 1

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書目名稱Antiarrhythmic Drugs影響因子(影響力)




書目名稱Antiarrhythmic Drugs影響因子(影響力)學(xué)科排名




書目名稱Antiarrhythmic Drugs網(wǎng)絡(luò)公開度




書目名稱Antiarrhythmic Drugs網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Antiarrhythmic Drugs被引頻次




書目名稱Antiarrhythmic Drugs被引頻次學(xué)科排名




書目名稱Antiarrhythmic Drugs年度引用




書目名稱Antiarrhythmic Drugs年度引用學(xué)科排名




書目名稱Antiarrhythmic Drugs讀者反饋




書目名稱Antiarrhythmic Drugs讀者反饋學(xué)科排名

Deduct

Die Physiologie der peripheren Nerven I type [2]. The fundamental question that should be answered is whether better results may be expected from the new approach of using drugs prolonging the action potential duration. Is this new hope justified?

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being about pharmacological treatment to a situation in which there is now marked uncertainty and general apprehension about the role of antiarrhythmic drugs. Until relatively recently the prevailing concept in antiarrhythmic therapy was that arrhythmias could be controlled by drugs which slowed con

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使閉塞

Jan Niklas Collet,Jan-Hendrik Herbstles at those site(s), may be an important tool to understand variability in response to antiarrhythmic drug therapy. Such understanding, in turn, may lead to an improved ability to predict patients at risk for serious adverse events, as well as to adjust doses to maximize efficacy.

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Interaction of Transient Ischemia with Antiarrhythmic Drugsic reflexes, as well as direct damage to the cell membrane, its constituent channels, pumps and gap junctions, and the supporting collagen matrix. Preexisting scarring due to myocardial infarction, cardiac hypertrophy, congestive heart failure, pulmonary disease and the like may contribute to the arrhythmogenic potential of transient ischemia.

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The Study of Pharmacokinetics and Pharmacodynamics as a Tool for Understanding Mechanisms of Antiarrles at those site(s), may be an important tool to understand variability in response to antiarrhythmic drug therapy. Such understanding, in turn, may lead to an improved ability to predict patients at risk for serious adverse events, as well as to adjust doses to maximize efficacy.

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Pharmacology of T and L Type Ca2+ Channels in Cardiac Tissueture describing the pharmacology of these two cardiac Ca channels is limited, particularly in reference to the pharmacology of the T type channel. Therefore, the emphasis here is to discuss the known pharmacology of both channels with particular attention to the T type Ca channel.

拍翅

A Shift from Class I to Class III Drugs in the Medical Treatment of Arrhythmias: Wishful Thinking? I type [2]. The fundamental question that should be answered is whether better results may be expected from the new approach of using drugs prolonging the action potential duration. Is this new hope justified?

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