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Titlebook: Animal Models of Neurological Disease, II; Metabolic Encephalop Alan A. Boulton,Glen B. Baker,Roger F. Butterworth Book 1992 Springer Scien

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21#
發(fā)表于 2025-3-25 04:06:07 | 只看該作者
Book 1992orders. Because of increasing public concern over the ethical treatment of animals in research, we felt it incumbent upon us to include this general preface to these volumes in order to indicate why we think further - search using animals is necessary and why animal models of psychiatric disorders,
22#
發(fā)表于 2025-3-25 08:02:56 | 只看該作者
Chemically Induced Models of Seizures,example, those that may result from a chemical imbalance, such as produced by hypoxia or hypoglycemia. Finally, in vivo seizure models will be described. Important information can be derived from in vitro or slice studies, but these models are beyond the scope of this chapter.
23#
發(fā)表于 2025-3-25 15:30:34 | 只看該作者
24#
發(fā)表于 2025-3-25 18:06:33 | 只看該作者
Alan A. Boulton,Glen B. Baker,Roger F. Butterworth
25#
發(fā)表于 2025-3-25 20:56:50 | 只看該作者
Book 1992 the animals outweighs the anticipated gains for human welfare, then the research is not ethical and should not be carried out. It is imperative that each individual researcher examine his/her own research from a critical moral standpoint - fore engaging in it, taking into consideration the animals’
26#
發(fā)表于 2025-3-26 01:58:24 | 只看該作者
27#
發(fā)表于 2025-3-26 07:02:13 | 只看該作者
Growth and Income Distribution in Iranne release impairs mental function, whereas the excessive release of dopamine and glutamate damages cells postsynaptically. Fundamental alterations in calcium homeostasis, particularly the ability of mitochondria to buffer calcium, appear to underlie these deficits. Furthermore, these changes in cal
28#
發(fā)表于 2025-3-26 10:23:46 | 只看該作者
https://doi.org/10.1007/978-3-642-51675-7s channels or simply reverses the direction of flow in the existing ones. These shunts permit toxins of intestinal origin to bypass the liver into the systemic circulation; hence the term “portal-systemic encephalopathy.”
29#
發(fā)表于 2025-3-26 14:57:28 | 只看該作者
30#
發(fā)表于 2025-3-26 20:19:49 | 只看該作者
https://doi.org/10.1007/978-3-642-57615-7n view of this, during progressive pyridoxine deficiency, the PLP-dependent enzymes are decreased to various extents, from no decrease to almost a complete depletion. The decreases in the activities of glutamic acid and 5-hydroxytryptophan decarboxylases during pyridoxine deficiency result in the re
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