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Titlebook: Animal Lectins: Form, Function and Clinical Applications; G. S. Gupta Book 2012 Springer-Verlag Wien 2012 animal lectins.lectins.Protein-L

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41#
發(fā)表于 2025-3-28 16:12:24 | 只看該作者
978-3-7091-4837-2Springer-Verlag Wien 2012
42#
發(fā)表于 2025-3-28 22:06:06 | 只看該作者
43#
發(fā)表于 2025-3-28 23:11:53 | 只看該作者
P-Type Lectins: Cation-Independent Mannose-6-Phosphate Reeptorss IGF-1 and -2, lacks kinase activity and is denoted as type II IGF receptor (IGF2R). The cell surface receptor for IGF2 also functions as a cation-independent M6PR. Therefore, cation-independent mannose 6-phosphate receptor (CI-MPR) is also referred as insulin-like growth factor 2 receptor (IGF2R)
44#
發(fā)表于 2025-3-29 05:14:31 | 只看該作者
Mannose-6-Phosphate Receptor Homologous Protein Familyotranslocation from the ER into the cytosol, and proteasomal degradation through ubiquitination. The quality-control system also surveys the ER lumen for terminally misfolded proteins. Polypeptides singled out by this system are ultimately degraded by the cytosolic ubiquitin-proteasome pathway. Key
45#
發(fā)表于 2025-3-29 10:32:52 | 只看該作者
Lectins of ERAD Pathway: F-Box Proteins and M-Type Lectinsigase and peptide:N-glycanase (PNGase) in the cytosolic ERAD pathway. Mannose trimming plays an important role by forming specific .glycans that permit the recognition and sorting of terminally misfolded conformers for ERAD. The EDEM (ER degradation enhancing α-mannosidase-like protein) subgroup of
46#
發(fā)表于 2025-3-29 14:56:01 | 只看該作者
47#
發(fā)表于 2025-3-29 17:46:56 | 只看該作者
Pentraxins: The L-Type Lectins and the C-Reactive Protein as a Cardiovascular Riskthe primary structure of the subunit, the pentraxins are divided into two groups: short pentraxins and long pentraxins. C-reactive protein (CRP), the first innate immunity receptor and serum amyloid P-component (SAP) are the two short pentraxins. Soluble pentraxins act as pattern recognition recepto
48#
發(fā)表于 2025-3-29 22:56:46 | 只看該作者
49#
發(fā)表于 2025-3-30 03:06:22 | 只看該作者
Galectin-1: Forms and Functionscentral nervous system after injury. The targeted overexpression of Gal-1 should be considered as a method of choice for the treatment of some kinds of inflammation-related diseases, neurodegenerative pathologies and muscular dystrophies. In contrast, the targeted inhibition of Gal-1 expression is w
50#
發(fā)表于 2025-3-30 08:00:30 | 只看該作者
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