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Titlebook: Angiogenesis in Brain Tumors; Matthias Kirsch,Peter McL. Black Book 2004 Springer Science+Business Media New York 2004 angiogenesis.brain

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發(fā)表于 2025-3-21 18:45:53 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱(chēng)Angiogenesis in Brain Tumors
影響因子2023Matthias Kirsch,Peter McL. Black
視頻videohttp://file.papertrans.cn/158/157413/157413.mp4
發(fā)行地址Offers an overview of the cellular, physiological and developmental basis of angiogenesis and an introduction into the basic science of this emerging field
學(xué)科分類(lèi)Cancer Treatment and Research
圖書(shū)封面Titlebook: Angiogenesis in Brain Tumors;  Matthias Kirsch,Peter McL. Black Book 2004 Springer Science+Business Media New York 2004 angiogenesis.brain
影響因子This volume "Angiogenesis in Brain Tumors" is part of the book series "Cancer Treatment and Research" and offers a detailed overview of the biology of angiogenesis in the central nervous system, the role of angiogenesis in brain tumor development and growth, and anti-angiogenic therapeutic applications. Our understanding of the mutual relationship between tumor growth and concomitant neovascularization has provided important insights into the biology of brain tumors and contributed to the development of new treatment strategies. Many of the early experimental findings on angiogenesis were made using brain tumor models. This was largely due to the fact that brain tumors are among the highest vascularized tumors. In addition, until to date all efforts to develop efficient therapies for these highly malignant tumors have met with little if any success . The scientific evidence on the role of angiogenesis in tumor development accumulated until today has already let to the initiation of several clinical trials. Despite the fact that not all expectations have been fulfilled, the results available so far are promising and suggest that anti- angiogenic therapeutic strategies may be of bene
Pindex Book 2004
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Enzyme Kinetic Data: Collection and Analysisels, while glioblastomas develop new vessels; 3) quantitation may have additional independent prognostic value over and above routine histologic grade in low-grade astrocytomas with low tumor cell proliferative indices. These findings have implications for the appropriateness of antiangiogenic therapies
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