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Titlebook: Angiogenesis; From the Molecular t Michael E. Maragoudakis Book 2000 The Editor(s) (if applicable) and The Author(s), under exclusive licen

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發(fā)表于 2025-3-21 19:02:35 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Angiogenesis
期刊簡(jiǎn)稱From the Molecular t
影響因子2023Michael E. Maragoudakis
視頻videohttp://file.papertrans.cn/158/157400/157400.mp4
學(xué)科分類Advances in Experimental Medicine and Biology
圖書封面Titlebook: Angiogenesis; From the Molecular t Michael E. Maragoudakis Book 2000 The Editor(s) (if applicable) and The Author(s), under exclusive licen
影響因子Proceedings of the 5th Biannual International Meeting onAngiogenesis: From the Molecular to Integrative Pharmacology, heldJuly 1-7, 1999, in Crete, Greece. .Angiogenesis, as a vastly complex biological process, has challengedresearchers from all basic scientific disciplines, includingpharmacology, biochemistry, physiology, embryology and anatomy. Thesignificance of this phenomenon for the study of disease states hasalso interested clinicians from a number of specialist fields. Thismultidisciplinary work reflects the growth of awareness of conceptssuch as angiogenesis based therapy, the enormous therapeutic andcommercial potential of which has attracted major research andinvestment in recent years. This volume, which aims to bridge the gapbetween basic and clinical methodology and understanding, presents themost up-to-date developments in this field.
Pindex Book 2000
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書目名稱Angiogenesis影響因子(影響力)




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Tie-1 Receptor Tyrosine Kinase Endodomain Interaction with SHP2: Potential Signalling Mechanisms andendodomain having a ligand-independent signalling role in the endothelial cell. The potential roles of ectodomain cleavage and cleavage activated signalling in regulating microvessel stability in angiogenesis, vessel remodelling and regression are considered.
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On the Mechanism(s) of Thrombin Induced Angiogenesisothelial cells increases the vectorial secretion of extracellular matrix proteins, a process essential at the final steps of angiogenesis. In addition, thrombin potentiates the VEGF-induced mitogenesis of endothelial cells. This can be explained by the upregulation of the VEGF receptors (KDR & flt-1
地板
發(fā)表于 2025-3-22 07:17:58 | 只看該作者
Endothelial Receptor Tyrosine Kinases involved in Blood Vessel Development and Tumor Angiogenesists of tissues, and is triggered by hypoxia. Deregulated angiogenesis plays an important role in the pathogenesis of a variety of diseases, including retinopathy and solid tumor growth (Folkman, 1995).
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Signal Transduction and Transcriptional Regulation of Angiogenesiss of these cell lines with a parental murine EC line, MSS31. The growth rate was almost identical among three cell lines. It appeared that gene expressions of matrix metalloproteinases (MMP-1, MMP-3, and MMP-9) as well as integrin beta3 were correlated with the level of Ets-1 expression. As a result
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Differential Contribution of Bradykinin Receptors in Angiogenesis inhibition in CVEC prevented BK/B1 induced cell growth. These results substantiate the protective and trophic role of BK on microvascular endothelium by demonstrating that Bl receptor stimulation on venular endothelium is mandatory for the angiogenic effect of BK through its coupling to NOS activat
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Sensitivity of Different Vascular Beds in the Eye to Neovascularization and Blood-Retinal Barrier Brvessels, and electron microscopy. Levels of transgene expression were determined by the polymerase chain reaction. In normal mice, retinal vessels are organized into a superficial and a deep capillary bed with some vessels forming a shunt between both beds. In a transgenic line of mice that overexpr
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