Titlebook: An Introduction to the Blood-Brain Barrier; Hugh Davson,Berislav Zlokovi?,Malcolm B. Segal Textbook 1993Latest edition The authors 1993 ce

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書(shū)目名稱An Introduction to the Blood-Brain Barrier影響因子(影響力)




書(shū)目名稱An Introduction to the Blood-Brain Barrier影響因子(影響力)學(xué)科排名




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書(shū)目名稱An Introduction to the Blood-Brain Barrier網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱An Introduction to the Blood-Brain Barrier被引頻次




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書(shū)目名稱An Introduction to the Blood-Brain Barrier讀者反饋學(xué)科排名

COLON

https://doi.org/10.1007/978-1-349-11882-3central nervous system; kinetics; morphology; research

NEG

https://doi.org/10.1007/978-3-662-38048-2 parenterally in the hope that they would attack infective organisms. Thus Ehrlich observed that many dyes, after intravenous injection, stained the tissues of practically the whole body, while the brain was spared. Later, Lewandowsky (1900) showed that the Prussian blue reagents (iron salt and pota

glowing

https://doi.org/10.1007/978-3-322-93596-0ince the central nervous tissue does not store glycogen to any great extent. Thus, the continuous metabolism of glucose must be adequately maintained by a continuous supply of the metabolite from the blood. Sugars, like amino acids, are highly water-soluble, and, being relatively large molecules, th

fluffy

https://doi.org/10.1007/978-3-322-93596-0to exist in the cell bodies, as well as in the axons and nerve terminals, of the central nervous system and, by definition, these are called . or .. Neuropeptides have been identified in different brain regions, and neuronal peptidergic pathways have been visualized and characterized in the central

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Der überlastete und passive Wohlfahrtsstaatt may be related to a disturbance of the barrier. The prerequisite for studies on barrier dysfunction is the establishment of suitable experimental animal models. Many, but far from all, models in experimental medicine are homologous, i.e. there is a correspondence in the aetiology of the disease an

fibula

Der überlastete und passive Wohlfahrtsstaatition, the cause of the condition in the model may be quite different from that in man. Finally, the model may have no resemblance to the disease but simply be a non-homologous or non-isomorphic representation that has value in either reflecting some aspects of the disease or for studying the therapy used for treating the disease.

使閉塞

Experimental Models in the Study of the Pathology of the Blood-Brain Barrier,ition, the cause of the condition in the model may be quite different from that in man. Finally, the model may have no resemblance to the disease but simply be a non-homologous or non-isomorphic representation that has value in either reflecting some aspects of the disease or for studying the therapy used for treating the disease.

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