Titlebook: Amyotrophic Lateral Sclerosis; Therapeutic, Psychol V. Cosi,Ann C. Kato,M. Poloni Book 1987 The Editor(s) (if applicable) and The Author(s)

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,Uranprovinzen und Uranvorr?te,While the article was in press, a new case with M-actyl-?- hexosaminidase B deficiency and progressive motor neuron disease has been reported by Hancock et al. (Biochem. Biophys. Res. Comm. 130:1185–1192, 1985).

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https://doi.org/10.1007/978-3-642-51409-8Amyotrophic lateral sclerosis (ALS) has been classified into sporadic and familial types[l]. The familial clustering does not imply a genetic mechanism but it seems a likely explanation in at least 5% of ALS cases.

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AddendumWhile the article was in press, a new case with M-actyl-?- hexosaminidase B deficiency and progressive motor neuron disease has been reported by Hancock et al. (Biochem. Biophys. Res. Comm. 130:1185–1192, 1985).

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The Pathogenetic Role of Metals in Motor Neuron Disease — The Participation of AluminumAmong possible causative factors, an association of metal(s) and motor neuron disease (MND) has been repeatedly reported since Aran’s description on lead intoxication with amyotrophy in 1850[1].

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https://doi.org/10.1007/978-3-7091-2193-1 that their cell bodies and dendrites are located within the central neurous system, whereas their axons constitute part of the peripheral nerves. The distal endings of the motor neurons in the motor end plates are unprotected by the blood-brain and blood-nerve barriers, and are therefore directly exposed to various substances in the blood.