Titlebook: Alcoholic/Non-Alcoholic Digestive Diseases; Hitoshi Yoshiji,Kosuke Kaji Book 2019 Springer Nature Singapore Pte Ltd. 2019 NAFLD.Non-Alcoho

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Durgesh Mishra,Xin She Yang,Dharm Singh Jatitus and its treatment have a significant impact on the clinical course of CLD. This review summarized the prevalence, diagnosis, and mechanisms of diabetes mellitus in patients with CLD. We also reviewed the clinical impact and therapeutic strategy for diabetes mellitus in patients with CLD. Recent

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Sunil Kumar Jangir,Naveen Hemrajaniers in a variety of tissues including the liver. Liver cancer most often occurs as hepatocellular carcinoma (HCC) complicating cirrhosis due to chronic viral infection, heavy alcohol consumption, or non-alcoholic steatohepatitis (NASH) which is a severe form of non-alcoholic fatty liver disease (NAF

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Daniel Pop,Gabriel Iuhasz,Dana Petcu in inducing oxidative stress. Mitochondria are the main site of cellular ROS production, and simultaneously have a well-organized antioxidant system. Therefore, mitochondria have evolved multiple systems of quality control to ensure that the requisite number of functional mitochondria is present to

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Daniel Pop,Gabriel Iuhasz,Dana Petcuase, hepatocyte apoptosis is frequently detected. Hepatocyte apoptosis is regulated by pro-apoptotic and anti-apoptotic bcl-2 family proteins. Among the anti-apoptotic proteins, Bcl-xL and Mcl-1 collaborate to prevent the activation of the mitochondrial apoptotic pathway and to maintain hepatocyte h

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Data Science and Big Data Computing, and the combination of genetic, environmental, and metabolic factors contributes to its development. Extensive research has been done to clarify the genetic factors. Candidate-gene approaches have focused on variants in the alcohol metabolizing enzymes (alcohol dehydrogenase 1B (.) and aldehyde de

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Alcohol-Induced DNA Injury in Esophageal Squamous Cell Carcinomaof DNA damage, plays a central role in alcohol-induced esophageal carcinogenesis. Acetaldehyde is mainly generated from the metabolism of ethanol by alcohol dehydrogenase 1B and is then detoxified to acetic acid by aldehyde dehydrogenase 2 (ALDH2). Alcohol consumption increases blood, saliva, and br

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Gastroesophageal Reflux Diseases and Lifestyle Factorsveral studies showed significant associations between GERD and lifestyle factors. In this chapter, we focused on obesity, metabolic syndrome, diabetes, hypertension, dyslipidemia, cigarette smoking, alcohol drinking, and late meals. We also discussed how these factors affect pathogenesis of GERD. We