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Titlebook: Alcohol and the Brain; Chronic Effects Ralph E. Tarter,David H. Thiel Book 1985 Springer Science+Business Media New York 1985 Nervous Syste

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發(fā)表于 2025-3-21 19:56:15 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Alcohol and the Brain
期刊簡(jiǎn)稱Chronic Effects
影響因子2023Ralph E. Tarter,David H. Thiel
視頻videohttp://file.papertrans.cn/153/152303/152303.mp4
圖書(shū)封面Titlebook: Alcohol and the Brain; Chronic Effects Ralph E. Tarter,David H. Thiel Book 1985 Springer Science+Business Media New York 1985 Nervous Syste
影響因子Chronic and excessive alcohol consumption disrupts a number of biologic sys- tems. Central nervous system pathology, associated with long-standing alcohol ingestion, has particularly deleterious consequences to the individual. Compro- mising brain functional integrity ultimately militates against psychosocial ad- justment, and this process is inevitably reflected as a substantial economic loss to society in the form of costs for providing medical and social services, as well as disability and absenteeism from work. This book marshals the literature pertinent to the effects of chronic alcohol abuse on brain structure and functioning. The material is divided into two parts: basic research and clinical issues. In the first section, the manifest neurologic consequences are described across the different levels of biologic organization, these being brain morphology, neurochemistry, neurophysiology, and neuro- psychology. In recognition of the multifactorial etiology of alcohol-related brain pathology, the influence and role of hepatic, endocrine, and nutritional factors are also examined. The second section addresses clinical syndromes and dis- orders. It will be noted that evidence acc
Pindex Book 1985
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Ethanol’s Action on Brain Biochemistryon and transmission. Using electrophysiologic techniques, one can directly assess the effects of ethanol on neuronal activity, and such studies have been performed (e.g., see reference 1). However, insights into the molecular mechanisms by which ethanol affects neuronal activity have, at present, be
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Nutrition and Alcoholismes will provide the reader with a background into specific methods used to evaluate nutritional status and the possible shortcomings of these techniques. Evidence for protein/calorie malnutrition will be provided in individual sections concerning alcoholics either with or without liver disease. Next
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Hepatocerebral Interactionshe development of inflammatory and fibrotic liver disease which, if it becomes severe enough, adversely affects brain function. The common name for this neurologic dysfunction associated with advanced liver disease is “hepatic coma.” The recognition and definition of this syndrome have a long and ta
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Neuropsychology of Alcoholismficiency, visuospatial integration, and learning capacity are reported frequently in such individuals.. Within the population of “social” drinkers, neuropsychologic capacity correlates negatively with alcohol consumption variables,. suggesting that the threshold for neurologic disruption may be lowe
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Biologic and Psychosocial Determinants in the Etiology of Alcoholism space—time continuum, a system is comprised of units with measurable relationships. In living systems, particles, atoms, and molecules, for example, are subsystems of the larger entities of cells, organs, and organisms of which they are comprised. The individual, his family, the community, and soci
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