Titlebook: Airways Smooth Muscle: Biochemical Control of Contraction and Relaxation; David Raeburn,Mark A. Giembycz (Lecturer) Book 1994 Springer Bas

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Cyclic Nucleotide Phosphodiesterases in Airways Smooth Muscle,etion, which are major features of this inflammation, probably contribute to the compromised airways function; however, it is generally agreed that the predominant cause of the airflow obstruction is a powerful and inappropriate constriction of bronchial smooth muscle as a consequence of exposure of

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Modulation of Agonist-Stimulated Phosphoinositide Turnover in Airways Smooth Muscle by Cyclic Nucleace receptors. Such agents may be released endogenously from neurones, may be blood-borne, or may be released by pro-inflammatory cells which infiltrate the airways under certain circumstances [1, 2]. Hyperreactive states of the airways are characterized by an increased smooth muscle tone and airway

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Airways Smooth Muscle: Biochemical Control of Contraction and Relaxation

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Airways Smooth Muscle: Biochemical Control of Contraction and Relaxation978-3-0348-7681-0

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Smooth Muscle Myosin: Molecule Conformation, Filament Assembly and Associated Regulatory Enzymes,le myosins share at least common major properties. In this review most of the information cited derives from studies on avian gizzard or pig stomach myosins but this should be readily applicable to myosin from airways smooth muscle.

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Phosphoinositidase C, Inositol Polyphosphates and Force Generation of Airways Smooth Muscle,ular Ca. in a manner that was not dependent on changes in membrane potential. It is now clear however, that a number of other membrane associated ‘phosphodiesterase-like’ enzymes including phospholipase D and phospholipase A. may also be involved in integrating the response of ASM cells to incoming