Titlebook: Aging and Recovery of Function in the Central Nervous System; Stephen W. Scheff Book 1984 Plenum Press, New York 1984 Alzheimer.age.aging.

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https://doi.org/10.1007/978-981-97-2740-7in the mammalian central nervous system (CNS). This research has taken on a very intriguing atmosphere since it has been suggested that this growth might serve as a basis for the recovery or restitution of function following damage. Conversely, one possibility must be recognized that aberrant neural

膠狀

https://doi.org/10.1007/978-981-97-2740-7ysiological systems also degenerate to varying degrees with advancing age, normal reproductive function ceases entirely and is replaced by some type of abnormal gonadal activity in the female (Aschheim, 1976). For example, in the aged rat two or three postreproductive syndromes are recognized includ

手段

Obstacles Facing the Stakeholders,recovery of function, has been observed since the relationship between the nervous system and behavior was first recognized (see Rosner, 1974). During the past decade, interest in this recovery process has been renewed by advances in our knowledge of neuronal plasticity in the mammalian CNS. Far fro

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https://doi.org/10.1007/978-981-97-2740-7n many reviews devoted to this question (Cotman, 1978; Finger, 1978a; Finger and Stein, 1982). Although the laboratory research has been exciting and there have been many new developments, clinicians have been slow to accept the idea that recovery after CNS injury is possible. Consequently, since li

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https://doi.org/10.1007/978-981-97-2740-7ervations showing that aphasia acquired after a child has learned speech is less severe and less permanent than in adults contributed to the widely held view that the young brain is more resilient to insults than the mature brain (cf. Guttman, 1942). However, this view has recently come under critic

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https://doi.org/10.1007/978-981-97-2740-7esses which progressively deplete brain and behavioral function. Age or disease-associated changes in behavior might be compensated or alleviated in a variety or combination of ways, however. These include behavioral-strategy alterations, pharmacologic manipulations, administration of growth or trop

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https://doi.org/10.1007/978-1-4613-2705-9Alzheimer; age; aging; anatomy; animals; behavior; biology; brain; cell; cortex; dementia; histology; neurobiolo

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Heresy

Longevity, Disease, and Autoimmune Reactions following Focal Cortical Injuries,ain can markedly shorten lifespan (Ordy .., 1967; Samorajski .., 1970; Rolsten .., 1973). But, on the other hand, the brain changes resulting from irradiation procedures are widespread and shed little light upon the contributions of different brain areas to aging and age-related pathology. Moreover,