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Titlebook: Aging Research in Yeast; Michael Breitenbach,S. Michal Jazwinski,Peter Laun Book 2012 Springer Science+Business Media B.V. 2012 Aging.Cell

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發(fā)表于 2025-3-25 07:04:45 | 只看該作者
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發(fā)表于 2025-3-25 11:31:10 | 只看該作者
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發(fā)表于 2025-3-25 13:17:03 | 只看該作者
https://doi.org/10.1007/978-3-030-28622-4ge that these reactive species cause, and to begin to dissect how these species may be involved in the ageing process. This chapter reviews the major sources of reactive oxygen species that occur in yeast cells, the damage they cause and how cells sense and respond to this damage.
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發(fā)表于 2025-3-25 18:51:15 | 只看該作者
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發(fā)表于 2025-3-25 20:12:56 | 只看該作者
Private Letters, Public Storiesalthy aging in mammals. We will focus on the mechanisms of life span regulation by the Tor/Sch9and the Ras/adenylatecyclase/PKA pathwayswith particular emphasis on those implicating age-dependent oxidativestressand DNA damage/repair .
26#
發(fā)表于 2025-3-26 01:14:10 | 只看該作者
https://doi.org/10.1057/9780230596542 chronological life spanhave allowed for the first unbiased screens aimed at directly identifying genes involved in determining yeast longevity. In this chapter we discuss large-scale life span studies performed in yeast and their implications for research related to the basic biology of aging.
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發(fā)表于 2025-3-26 05:03:36 | 只看該作者
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發(fā)表于 2025-3-26 15:56:27 | 只看該作者
Book 2012isms provide models for aging research that are relevant to organismic aging and to the aging processes occurring in the human body. Replicative aging, in which only the mother cell ages while the daughter cell resets the clock to zero is a model for the aging of stem cell populations in humans, whi
30#
發(fā)表于 2025-3-26 20:44:40 | 只看該作者
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