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Titlebook: Aging 2000: Our Health Care Destiny; Volume 1: Biomedical Charles M. Gaitz (Head, Gerontolgy Center Assistan Conference proceedings 1985 Sp

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發(fā)表于 2025-3-21 19:22:19 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Aging 2000: Our Health Care Destiny
期刊簡稱Volume 1: Biomedical
影響因子2023Charles M. Gaitz (Head, Gerontolgy Center Assistan
視頻videohttp://file.papertrans.cn/152/151445/151445.mp4
圖書封面Titlebook: Aging 2000: Our Health Care Destiny; Volume 1: Biomedical Charles M. Gaitz (Head, Gerontolgy Center Assistan Conference proceedings 1985 Sp
影響因子The seventeenth annual symposium sponsored by the Texas Research Institute of Mental Sciences was held October 23-26, 1983, in Houston, Texas. The theme was Aging 2000: Our Health Care Destiny. This volume on biomedical aspects and a companion volume on psycho- social aspects include edited versions of the presentations by about 80 speakers. Their papers were directed at practitioners, researchers, and medical educators who will be active and productive in the year 2000, and we focused on those who would influence the evolution of care of elderly persons during the next 17 years. We chose topics that would be of particular interest to teachers and current planners in the disciplines concerned with delivery of health and social services. We believe that having a core of more qualified and better trained practitioners will help the population of aged persons achieve a higher level of physical and mental health, life satisfaction and happiness, to find better coping techniques and control of environmental stresses, and to attain personal and social goals. Our Goals While preparing for the symposium we knew that the status of the art in 1983 obviously would be the base from which we wo
Pindex Conference proceedings 1985
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W. B. Harrison,B. A. C. DijkmansRecent findings from this lab include the progressive decrease during normal human aging of caudate D. receptors and cortical S. receptors. Also, the ovary-dependent neuroendocrine aging syndrome of female rodents is described.
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A Progress Report on Neurochemical and Neuroendocrine Regulation in Normal and Pathologic AgingRecent findings from this lab include the progressive decrease during normal human aging of caudate D. receptors and cortical S. receptors. Also, the ovary-dependent neuroendocrine aging syndrome of female rodents is described.
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C. Darras,C. Bourdy,G. Verriestging and the aged will be influenced by socioeconomic factors and by the status and trends of the health and disability characteristics of the elderly population. The increase in longevity has not been accompanied by a significant change in the onset of disability, namely, the compression of infirmi
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Nicole T. Tobin,Lisa M. Frenkel by the basic nature of human biology, and is similar for a wide variety of different physiological systems as well as for the onset pattern of the major degenerative diseases. Efforts to modify the aging pattern will have consequences in terms of the quality of later life which are not independent
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Erik D. A. Clercq,Anne-Mieke I. Vandamme span, food restriction also retards age-related physiological deterioration and age-related disease processes. Food restriction started in adult life is as effective as food restriction started in early life in regard to these effects. Restriction of protein but not calories did increase the median
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Joffroy Beauquier,Loys Thimonierage-dependent increase in soluble hexokinase activity and a significant decrease in activity of the key glycolytic enzyme phosphofructokinase. This decrease in the glycolytic turnover capacity lowers acetyl coenzyme A, necessary for the biosynthesis of acetylcholine. Therefore normal aging of the br
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