Titlebook: Advances in Multiple Sclerosis and Experimental Demyelinating Diseases; Moses Rodriguez Book 2008 The Editor(s) (if applicable) and The Au

上釉彩

https://doi.org/10.1007/BFb0011119 injury and promotes maximal remyelination within 5 weeks after a single low dose (25 μg/kg). The IgM isoform of this reparative antibody is required for in vivo function. We hypothesize that the IgM clusters membrane domains and associated signaling molecules on the surface of target cells. Current

pulmonary

https://doi.org/10.1007/BFb0011119 axonal damage, and remyelination, and studies utilizing in vivo T1 and T2 relaxometry, provide insight into the pathology of demyelinating diseases at previously unprecedented details. The technical challenges of small voxel in vivo MR spectroscopy and the biologically relevant information obtained

motor-unit

廚房里面

0070-217X etic tools for identifying patients who are most likely to experience spontaneous remyelination. Epidemiology studies offer additional avenues of treatment. Exa978-3-642-43783-0978-3-540-73677-6Series ISSN 0070-217X Series E-ISSN 2196-9965

NEG

清醒

Obscure

ASSAY

CLAY

Advances in Multiple Sclerosis and Experimental Demyelinating Diseases

委派

Benign Multiple Sclerosis: A Distinct Clinical Entity with Therapeutic Implications,clerosis. Based on the Olmsted County population prevalence cohort study performed at the Mayo Clinic, MS patients with EDSS scores of 2 or lower followed for a period of 5–10 years have a very small risk of developing disability over the next 10–20 years. Based on these findings, this chapter revie