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Titlebook: Advances in Research on Neurodegeneration; Volume 7 Y. Mizuno,D. B. Calne,M. B. H. Youdim Conference proceedings 2000 Springer-Verlag Wien

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31#
發(fā)表于 2025-3-26 21:32:29 | 只看該作者
The myelin oligodendrocyte glycoprotein (MOG): a model for antibody-mediated demyelination in expersingly, recent data suggest that this autoimmune response is in part regulated by polymorphisms in the MOG gene itself, an observation that may have important implications for the genetic and immunological stratification of patients with multiple sclerosis.
32#
發(fā)表于 2025-3-27 03:44:38 | 只看該作者
33#
發(fā)表于 2025-3-27 06:17:10 | 只看該作者
David T. Melnekoff,Alessandro Laganàsingly, recent data suggest that this autoimmune response is in part regulated by polymorphisms in the MOG gene itself, an observation that may have important implications for the genetic and immunological stratification of patients with multiple sclerosis.
34#
發(fā)表于 2025-3-27 10:24:12 | 只看該作者
refecture in Japan on January 20 to 22, 1999. Karuizawa is a nice summer as well as winter resort close to the place for winter Olympic game in the year of 1998. Over 40 scientists gathered together and made hot discussion. Neurodegeneration and neuroinflammation are two major and important neurolog
35#
發(fā)表于 2025-3-27 17:37:00 | 只看該作者
36#
發(fā)表于 2025-3-27 20:45:07 | 只看該作者
PARKIN as a pathogenic gene for autosomal recessive juvenile parkinsonism,ethnic origins, providing a definitive evidence for the Parkin to be a causative gene for AR-JP. The predicted structure of Parkin protein and its mutation provide important clues for studying the functional role of the Parkin protein in leading to selective degeneration of nigral neurons in the brains of AR-JP patients.
37#
發(fā)表于 2025-3-27 22:52:22 | 只看該作者
https://doi.org/10.1007/978-3-7091-6284-2Alzheimer‘s Disease; Multiple Sclerosis; Neurodegeneration; Neuroinflammation; Parkinson; Parkinson‘s Dis
38#
發(fā)表于 2025-3-28 03:41:56 | 只看該作者
39#
發(fā)表于 2025-3-28 08:30:38 | 只看該作者
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40#
發(fā)表于 2025-3-28 11:33:17 | 只看該作者
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