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Titlebook: GeNeDis 2018; Genetics and Neurode Panayiotis Vlamos Conference proceedings 2020 The Editor(s) (if applicable) and The Author(s), under exc

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發(fā)表于 2025-3-21 18:05:12 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱GeNeDis 2018
副標(biāo)題Genetics and Neurode
編輯Panayiotis Vlamos
視頻videohttp://file.papertrans.cn/381/380994/380994.mp4
概述New developments in research on mental health and aging.This volume focuses on genetics and neurodegeneration.Based on the 3rd World Congress on Genetics, Geriatrics, and Neurodegenerative Disease Res
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: GeNeDis 2018; Genetics and Neurode Panayiotis Vlamos Conference proceedings 2020 The Editor(s) (if applicable) and The Author(s), under exc
描述The 3rd World Congress on Genetics, Geriatrics, and Neurodegenerative Disease Research (GeNeDis 2018), focuses on recent advances in genetics, geriatrics, and neurodegeneration, ranging from basic science to clinical and pharmaceutical developments. It also provides an international forum for the latest scientific discoveries, medical practices, and care initiatives. Advanced information technologies are discussed, including the basic research, implementation of medico-social policies, and the European and global issues in the funding of long-term care for elderly people.?.
出版日期Conference proceedings 2020
關(guān)鍵詞neurodegenerative diseases; geriatrics; mental health; brain disorders; memory
版次1
doihttps://doi.org/10.1007/978-3-030-32633-3
isbn_softcover978-3-030-32635-7
isbn_ebook978-3-030-32633-3Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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Uoldelul Chelati Dirar,Karin Pallaveressful therapeutically even though they have been associated with reductions in amyloid. Currently, efforts are underway to deliver these therapies to individuals with very early symptoms or at risk for Alzheimer’s dementia by virtue of genetics or a brain amyloid PET scan. Results from these studie
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Geriatric Pharmacology Overviewthe neural lineage causes uncontrolled increase in autophagy. In turn, increased autophagy mediates the synaptic defects caused by BDNF deficiency. Thus, fasting suppresses autophagy in regions of the mouse forebrain, thereby promoting synaptic remodeling and memory through a BDNF-regulated mechanis
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Future Action and Ageing Preparation,lications is also an area under heavy research but with mixed results at the moment. While brain training with scientific techniques can improve performance in specific areas, appropriate physical exercises, as well as proper nutrition, have additional benefits for the body. Supplementary studies wi
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Molecular and Cellular Basis of Aging,to high phenol concentration. The link of a polyphenol found in extra virgin olive oil, namely, tyrosol, with the protein tyrosinase, associated to Parkinson’s disease is underlined as a paradigm of affiliation between polyphenols and neurodegenerative disorders.
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Renupama Bhoi,Arpita Patro,Angana Sarkar-like structures (“organoids”) mimicking native brains. Three-dimensional (3D) systems, called cerebral organoids, can recapitulate distinct architectures of the human brain, such as fluid-filled cavities resembling brain ventricles and tissues organized in layers including progenitor ventricular an
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Clinical Examination of the Airwayschanges, including acetylcholine deficiency, glutamate excitotoxicity, extracellular deposition of β-amyloid (Aβ plaques), intracellular neurofibrillary tangles by hyperphosphorylated tau protein deposits, neuroinflammation, and widespread neuronal loss (Godyn et al. Pharmacol Rep 68:127–138, 2016;
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Vera Kutzinski,Ingo Schwarz,Ottmar Etteavailability of these supports to psychiatry trainees and whether their lifestyles allow them time to spend on these activities. Though it is impossible to remove all stressors from psychiatry, interventions to improve trainee wellbeing must consider factors that improve resilience and support as we
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